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0.3854 | 2a9a962664d6413eb5e53a7f286ab040 | Isotherms of the temperature T(z,t) generated in the brake disc along the axial direction z over time of braking applications: (a) no. 1; (b) no. 2. | PMC9571107 | materials-15-06821-g009.jpg |
0.488014 | 558b62d1d700438d8f9a188c4da3c2ba | Distributions of the temperature T along the depth of the disc z, at the selected time moments t within the braking processes: (a) no. 1; (b) no. 2. | PMC9571107 | materials-15-06821-g010.jpg |
0.411395 | ca6baf8a88e04c1eac3dbcb155193134 | A simplified diagram of complex plant responses to stressful environmental stimuli: oxidative burst and its consequences as a universal reaction to different stressors are shown, as well as stressor-dependent reactions leading to plant growth retardation and a decline in productivity. | PMC9571535 | plants-11-02544-g001.jpg |
0.402735 | 487ea862ac0445fb9bacef56b94f0808 | Types of exogenously applied priming agents discussed in this review, as well as their possible applications and the beneficial consequences of treatments that protect plant cells and provide improved defense potential. | PMC9571535 | plants-11-02544-g002.jpg |
0.489979 | 7d5d99445f824ff7ab881b65791cf490 | Image showing the Albus Home at the bedside (left) and the reference polygraphy device using thoraco-abdominal respiratory effort bands (right). For illustration, duvets were removed to show the device, but participants slept using own duvets under usual sleeping conditions. | PMC9573065 | sensors-22-07142-g001.jpg |
0.441001 | 3061e2621c4e4fc78a908b95611d6412 | (a) Scatter plot showing correlation between RR readings between Albus system and reference (clinician-counted polygraphy). (b) Bland–Altman plot showing agreement in RR readings between Albus system and reference (solid line shows mean difference of −0.38 breaths/min and dotted lines show 95% limits of agreement from 0.8 to −1.6 breaths/min). There are, in total, 900 comparisons across 32 participants, with each point depicting a RR reading for a 30-s period. | PMC9573065 | sensors-22-07142-g002.jpg |
0.415394 | 78ed729bde674a6eaea96d5dc302daae | (a) Scatter plot showing correlation between hourly mean RR readings between Albus system and reference (clinician-counted polygraphy). (b) Bland–Altman plot showing agreement in hourly mean RR readings between Albus system and reference (solid line shows mean difference of −0.46 breaths/min and dotted lines show 95% limits of agreement from 0.04 to −0.88 breaths/min). There are, in total, 20 comparisons across 10 participants, with each point depicting a mean RR reading for a 1-h period. | PMC9573065 | sensors-22-07142-g003.jpg |
0.452243 | 7e74e8a4727d42e79bddd9a030804bf6 | The shunt active filter environment. | PMC9573495 | sensors-22-07516-g001.jpg |
0.35212 | 34223f114a614f27bda8dd3476fdf75b | A single-phase circuit of the LCL output filter. | PMC9573495 | sensors-22-07516-g002.jpg |
0.434924 | 40790cf3691449fcaa3fc5b62c73a134 | Advanced PLL-based generalized instantaneous power algorithm. | PMC9573495 | sensors-22-07516-g003.jpg |
0.460133 | 567a8348fe754682b6b3bce2d3b1a4c3 | Flowchart of the C-HOSM controller. | PMC9573495 | sensors-22-07516-g004.jpg |
0.475443 | 4366e6094eed4742a52fd381e04eb000 | Tracking and controls for the SMC, C-HOSM, 2-SMC Twisting, and Super-Twisting algorithms. | PMC9573495 | sensors-22-07516-g005.jpg |
0.414485 | 30ac3e25337a41debc7d3518e0d1c913 | Spectrum analysis of network current before filtration. | PMC9573495 | sensors-22-07516-g006.jpg |
0.444543 | d3953687c1eb4fe7856239f929dedb42 | Current and voltage filtering using time domain SMC. | PMC9573495 | sensors-22-07516-g007.jpg |
0.461962 | f18c8a10f9bc4b34bef61d9ab06433ab | Current filtering using the C-HOSM controller. | PMC9573495 | sensors-22-07516-g008.jpg |
0.40741 | de372d65228f41f9b921aa9fb383450a | Current filtering using the twisting-controller. | PMC9573495 | sensors-22-07516-g009.jpg |
0.423879 | afbd1c6cc3be441e9b6f84fc3509ed20 | Current filtering using the super-twisting controller. | PMC9573495 | sensors-22-07516-g010.jpg |
0.363152 | 169c55e7659d4126ad539a7e97a1b32f | DC voltage using the C-HOSM, twisting, and super-twisting controllers. | PMC9573495 | sensors-22-07516-g011.jpg |
0.469043 | 127b74a8f98f4cff966a0c280f69900b | Energy balance of the SAF environment. | PMC9573495 | sensors-22-07516-g012.jpg |
0.432202 | aac6d4f87baa44aa90fa40f40d98ecdb | Experimental bench of the generalized identification method. | PMC9573495 | sensors-22-07516-g013.jpg |
0.43035 | e54305a03b324b49bad44a1111be67ee | Experimental results of the three-phase grid voltage and positive sequence voltage. | PMC9573495 | sensors-22-07516-g014.jpg |
0.377807 | 7f6d746a4e9b4ee4a5298c2dfe82d4c7 | (a,b). Experimental results of the load current, distorted identified current, and grid current for two load levels (4 and 2.2 kW). | PMC9573495 | sensors-22-07516-g015.jpg |
0.44268 | f2bbb3eaa36c40e89681c64f338dad04 | Electrical grid of the textile factory. | PMC9573495 | sensors-22-07516-g016.jpg |
0.419761 | 3edd99d1222b4684ad30e25b6c51071a | Power quality phenomena measurements using C.A 8334B. | PMC9573495 | sensors-22-07516-g017.jpg |
0.44324 | c0dc7c1799bc485593999cf3aa248d78 | Real measurements of fundamental, harmonic currents, and voltage ratios and power factor using the power analyzer: (a) load current, (b) load current THD, (c) grid voltage THD, (d) power factor, (e) current harmonic distortion (rank 5), and (f) current harmonic distortion (rank 7). | PMC9573495 | sensors-22-07516-g018.jpg |
0.44959 | d236ad03e6a84983af41031bc4257997 | Current filtration: C-HOSM controller. | PMC9573495 | sensors-22-07516-g019.jpg |
0.451294 | c39bb64815664fbb9c6c19d03957ed2f | Current filtration: 2-SMC Twisting controller. | PMC9573495 | sensors-22-07516-g020.jpg |
0.448937 | 1f482fa4b832495c81f38b0063e8109c | Current filtration: 2-SMC Super-Twisting controller. | PMC9573495 | sensors-22-07516-g021.jpg |
0.505922 | 823581201e3c46159f730b2158c4cb91 | Reported elements (with color background) that can form XenesBlue background, metals; green background, metalloids; yellow background, nonmetals. White background, unreported yet. | PMC9573930 | gr1.jpg |
0.426903 | 47f645549b83460288c8ae4e44d3387d | Brief synthesis process and structure of hydrogel and Xene, respectively(A) Traditional hydrogels are fabricated by polymers and water via a process called gelation. Crosslinkers can be used to connect polymer chains. And covalent and noncovalent bonds also contribute to the 3D mesh development.(B) Xenes can be synthesized via two methods. The top-down method always requires a layered structure of bulk materials and uses techniques to break the interlayer forces. The bottom-up method takes no account of the bulk structure, synthesizing Xenes from their precursors. Most Xenes have a two-dimensional honeycomb lattice. | PMC9573930 | gr2.jpg |
0.420267 | e2f29f00241d42ba9eb02f25b4074fb8 | Synthesis of Xene hydrogels via three different methods(A) Xenes incorporation in crosslinked hydrogels, (B) hydrogel crosslinking in the presence of Xenes, and (C) self-gel of Xenes. | PMC9573930 | gr3.jpg |
0.451502 | 58a3c360c9f544c8be700fcfc30a58ac | Properties of hydrogel, Xene, and Xene hydrogel | PMC9573930 | gr4.jpg |
0.428857 | 162e5686a7114994967f19000dd9b981 | Medical applications of Xene hydrogels(A) Loaded with therapeutic drugs, nanoscale Xene hydrogel can be delivered to the tumor site and be decomposed in tumor cells upon light irradiation to release the cargos. Xenes can function as photothermal agents against cancer cells.(B) Xene hydrogel can be utilized in cardiac tissue replacement, bone generation, and neural-like differentiation.(C) Xene hydrogel can be applied to protect wounds from infectious microorganisms. Similar to the case in the tumor site, Xene can convert light energy into heat to kill focal bacteria. Also, released cargos can exert different functions such as promoting angiogenesis. | PMC9573930 | gr5.jpg |
0.43021 | 7e7caffbc3bd4a6cb8b42af8d0740bb4 | Application of Xene hydrogel in biomedicine and biosensor(A) Scheme of in situ sprayed BP gel for accelerated wound healing in diabetic ulceration.(B) Pseudocolor scanning electron microscopy (SEM) image of BP@Gel.(C) Images of wound healing in mice skin at different times after different treatments: G1, control; G2, Gel; G3, BP@Gel; and G4, BP@Gel@Lid + NIR.156(D) Schematic illustration of the incorporated dual-modality Cys-RGO hydrogel microfluidic biosensor chip for the detection of cMb.(E) A photograph of the nanoengineered integrated dual-modality mesoporous Cys-RGO hydrogel microfluidic biosensor chip.(F) SEM image of the integrated Cys-RGO hydrogel.(G) Incorporated dual-modality Cys-RGO hydrogel sensor mechanism revealing the coupling of light and voltage sources to measure electrochemical and SPR signals157 (copyright Proceedings of the National Academy of Sciences of the United States of America, 2020; copyright Royal Society of Chemistry, 2020). | PMC9573930 | gr6.jpg |
0.432145 | 60990a178c68482d9d0cb639601c089f | Lithium storage mechanism of 2D-TiO2-2D heterostructured electrode and PANI/GO hybrid hydrogels formation, shaping/reduction process, and their conductivity(A) Schematic illustration on possible diffusion and transfer paths of Liþ ions in BPNs@TiO2@G composite electrode, (B) C 1s and (C) Li 1s spectra of BPNs@TiO2@G electrode after cycles. Inset in (C) shows the crystal structures of Li3P.65 (D) Schematic illustration of PANI/GO hybrid hydrogels formation and further shaping/reduction process; photos of the self-assembly of PANI/GO hybrid hydrogels and the reduction of PANI/RGO (the inset) as well as the corresponding SEM and TEM images of samples after freeze-drying158 (copyright Elsevier, 2019; copyright John Wiley and Sons, 2018.) | PMC9573930 | gr7.jpg |
0.44642 | 5330fe8a39234ef59aad3ffa3f9495bc | Application of Xene hydrogel in water treatment(A) Fabrication process of the sandwich-structured filter systems and the description of bacteria removal–disinfection.(B) Removal efficiency for E. coli treated with the F-paper and CSP with different crosslinking degrees.43(C) Thermographic image of CSBPP4 after 10 min of NIR illumination.(D) Schematic illustration of the synthesis process and catalytic performances of the prepared rGO-based composite hydrogels.(E) Schematic of the Langmuir-Hinshelwood model and (F) the catalytic mechanism for 4-NP catalytic reduction160 (copyright Elsevier, 2020; copyright Royal Society of Chemistry, 2019). | PMC9573930 | gr8.jpg |
0.444541 | 1e87df6b2e46448d8829719dd3cc5191 | A subungual mixed tumor in a 65-year-old female.A. On anteroposterior and lateral radiographs of the left index finger, scalloping (arrow) is observed on the dorsal side of the left index finger. No periosteal reaction, cortical destruction, and tumor matrix ossification are observed.B. The tumor (arrows) shows hyperintensity on sagittal T2-weighted image (left) and fat-suppressed T2-weighted image (right) (upper image). The tumor (arrows) shows isointensity on axial T1-weighted image (left lower image) and homogeneous enhancement on axial contrast-enhanced T1-weighted image (right lower image).C. An axial dynamic contrast enhancement MR image (left) is displayed, and the circular ROI is drawn on the tumor; the corresponding time-intensity curve (right) shows a rapid initial enhancement followed by sustained late enhancement, suggesting a benign lesion.D. Intraoperatively, a 3 × 3 mm2-sized whitish mass is noted in the subungual space of the left index finger.E. On microscopy, the lesion is well circumscribed (hematoxylin & eosin stain; × 40) (left); the tumor is composed of variable myoepithelial cells (open arrow) and myxoid stromal components (arrow) in a mixture of patterns (hematoxylin & eosin stain; × 100) (right).ROI = regions of interest, SD = standard deviation | PMC9574269 | jksr-83-1134-g001.jpg |
0.561101 | 7e83c0e2eec14efe8fcc72459986cc1b | Histograms for UMN signs of the upper (A) and lower limb (B). Numbers of limbs are presented according to disease (ALS or MSA) and severity in the UMN for the upper limb (A) and lower limb (B). | PMC9574772 | gr1.jpg |
0.595357 | 0e0f667cecb341d6a245720312305ac4 | Histograms for results of CMCT. Numbers of limbs are presented according to disease (ALS or MSA) and results of the CMCT testing for the upper limb (A) and lower limb (B). | PMC9574772 | gr2.jpg |
0.37347 | 7d3f07b9f8b54b419f1ef7176dec17e8 | Relation between CMCT and UMN sign. CMCT values are shown according to disease (ALS and MSA) and UMN sign (none, mild, and severe). Each dot represents a limb: (A) FDI-CMCT, (B) TA-CMCT. | PMC9574772 | gr3.jpg |
0.473202 | 0d913be408db446a897b546686c58a0b | Results of distortion product otoacoustic emissions tests before (day 1) and after (day 6) noise exposure. Control, trauma, trauma+berberine, and berberine groups mean the results of distortion product otoacoustic emissions of these groups, performed on day 6. | PMC9575033 | 1806-9282-ramb-68-09-1330-gf01.jpg |
0.474303 | 63f76ea698374b2bbf84717a5dcb1d66 | Histopathological appearance of cochlear tissues. (A) Acoustic Trauma Group. Degeneration and necrosis in ganglia (arrow heads), desquamation, and severe decrease in number of outer hair cells (arrows). (B) Control Group. Normal histological appearance. (C) Berberine Group. Normal histological appearance. (D) Acoustic Trauma+Berberine Group. Degeneration in ganglia (arrow heads), mild desquamation, and decrease in number of outer hair cells (arrows). Hematoxilen & eosine, Bar: 100 μm. Immunohistochemical appearance of cochlear tissues. (E) Acoustic Trauma Group. Severe cytoplasmic 8-hydroxy-2-deoxyguanosine expression in ganglia and outer hair cells (arrow heads). (F) Control Group. Negative 8-hydroxy-2-deoxyguanosine expression. (G) Berberine Group. Negative 8-hydroxy-2-deoxyguanosine expression. (H) Acoustic Trauma+Berberine Group. Mild 8-hydroxy-2-deoxyguanosine expression in ganglia and outer hair cells (arrow heads). Immunohistochemistry-peroxidase, Bar: 100 μm. | PMC9575033 | 1806-9282-ramb-68-09-1330-gf02.jpg |
0.440877 | 46e2e775a69f4eacbf228746d69ca864 | Flowchart of the patient included in the study. FM, Foundation Medicine. | PMC9575317 | fonc-12-910117-g001.jpg |
0.442552 | 1a312e0dda0a41279442ff5ccd7c18f0 | Comprehensive visualization of the genomic landscape of NSCLC. (A) young (B) older patients. | PMC9575317 | fonc-12-910117-g002.jpg |
0.528574 | 33e980c598814e4f8221b95b78e53cb2 | Comparative analysis of mutation between age groups in genes more frequently altered (A). Comparison between age groups of the structural alteration in p53 (B) and in EGFR (C). | PMC9575317 | fonc-12-910117-g003.jpg |
0.512685 | 373d692a8a804ffcbfb159d3f219c66f | TMB comparison between younger and older patients with NSCLC (A). (B) Distribution of TMB groups between younger with older patients (B). | PMC9575317 | fonc-12-910117-g004.jpg |
0.398591 | 0c48e767b73d4d348d2cc7ffaffa361a | Copy number variation in older vs younger patients. | PMC9575317 | fonc-12-910117-g005.jpg |
0.473726 | 7573d20df01b455780da57d45d695e12 | Micrographs of ANME-1 and partner bacteria of the Loki's Castle barite field. (A) Aggregates of ANME-1 (ANME-1–350 probe) and Deltaproteobacteria (Delta495 probes) in Loki's Castle barite field sediments and (B) barite chimneys. ANME-1 and Deltaproteobacteria are in red and green, respectively. (C) Filaments of ANME-1 in the upper section of a barite chimney, stained in green. Scale bars are reported for A and B-C. | PMC9576274 | fiac117fig1.jpg |
0.427039 | 8292e016d0ae4dfbadb3896784ee8175 | Phylogenomic analysis of ANME-1. (A) Phylogenomic tree of the ANME-1 order based on concatenated alignment of 35 marker genes. The ANI-defined AMOR subgroups are highlighted by colors as in the legend. For each genome, the environment of origin is indicated in parenthesis next to the leaf name. The genus- and family- level classification from GTDB-tk is indicated on the right (g_ for genus; f_ for family). Bootstrap values < 60 are in red. (B) Relative abundance of the AMOR subgroups in various samples at Loki's Castle vent field and Jan Mayen vent field. The estimated temperature at each site is indicated by symbols. ‘LCBF’: Loki's Castle barite field, ‘JMVF’: Jan Mayen vent field, ‘LCVF’: Loki's Castle vent field. | PMC9576274 | fiac117fig2.jpg |
0.435056 | 840507fb12b54551ab76847fb200980c | Gene clusters enriched in generalist ANME-1 and their cellular function. (A) Pangenome of the ANME-1 order. Hierarchical clustering is expressed by the dendrogram on the left of the phylogram. Genomes are thereafter divided into generalist and vent-specific. The core and the accessory pangenome are indicated. The coverage of COG, KOfam and Pfam annotations and the number of genomes contributing to each gene cluster are given below the phylogram. GTDB-tk classification is on the right. (B) Diagram of the cellular function of 32 genes enriched in generalist ANME-1. The occurrence of each gene in generalists and vent-specific genomes is indicated in the heatmap. More details of each cellular function are given in Table S8. | PMC9576274 | fiac117fig3.jpg |
0.399185 | 19628f8348984795b8d6b85d0cdad913 | Effects of ischemic stroke and exercise preconditioning on neurological scores and infarction volume. (a) The neurological function scores of each group of mice were assessed at 24 h (n = 12). (b) Comparison of cerebral infarct volume of the ipsilateral brain between groups (n = 4). (c) Infarct size was determined by tetrazolium chloride (TTC) staining after cerebral ischemia-reperfusion (n = 4), and the infarct area was identified by nonstaining region, while the live area should turn red. ∗∗P < 0.01 vs. Sham; ##P < 0.01 vs. MCAO; ^^P < 0.01 vs. EP+Sham. | PMC9576414 | MI2022-2124230.001.jpg |
0.597498 | c37e9a7185524fc389630386337b77d5 | Effects of ischemic stroke and exercise preconditioning on the cognitive function of mice. (a) Discrimination index of mice in novel object recognition task (n = 7). (b) Spontaneous alternation rate of mice in Y-maze test of spontaneous alternation (n = 7). ∗∗P < 0.01 vs. Sham; #P < 0.05 vs. MCAO. | PMC9576414 | MI2022-2124230.002.jpg |
0.420949 | ca7049d830454814845ba0b9c352f0ba | Effects of ischemic stroke and exercise preconditioning on the histopathological morphology of the hippocampal region (400x). | PMC9576414 | MI2022-2124230.003.jpg |
0.523367 | 95c789b9938a45d0beaec9ffea03276c | Effects of ischemic stroke and exercise preconditioning on proinflammatory cytokines in serum of each group. (a) The expression of IL-1β in brain tissue of mice (n = 8). (b) The expression of IL-18 in brain tissue of mice (n = 8). ∗∗P < 0.01 vs. Sham; #P < 0.05 vs. MCAO; ^P < 0.05 vs. EP+Sham. | PMC9576414 | MI2022-2124230.004.jpg |
0.500865 | 4d56ffbc160e4ff6a583658a26fdf30b | Representative western blots (a) and quantification data of NLRP3 (b), Caspase-1 (c), IL-1β (d), and IL-18 (e) for each group (n = 5). ∗∗P < 0.01 vs. Sham; #P < 0.05 vs. MCAO. | PMC9576414 | MI2022-2124230.005.jpg |
0.39447 | c7dabe20d6b244c98f38d7396ede6245 | Ischemic stroke and exercise preconditioning affect the alpha and beta diversity of gut microbiota in mice (n = 6). (a) Chao1; (b) Faith's PD; (c) PCoA analysis. | PMC9576414 | MI2022-2124230.006.jpg |
0.453091 | 08878a05f8c041ab9a3f5dd61f93bd65 | Phylum-level effects of ischemic stroke and exercise preconditioning on gut microbiota (n = 6). (a) The abundance of gut microbiota at the level of phylum. (b) Interactive presentation of sample taxonomic composition at the level of phylum. | PMC9576414 | MI2022-2124230.007.jpg |
0.399869 | fe67a9b0b8ae475086f0969ad8733d4c | Effects of ischemic stroke and exercise preconditioning on the abundance of gut microbiota at the genus level (n = 6). (a) The abundance of gut microbiota at the genus level. (b) Log2 values of the multiple of ASV (fold change; FC) compared to the MCAO group in the Sham group, with positive values indicating upregulation, while negative values indicate downregulation. (c) Log2 values of the multiple of ASV (fold change; FC) in the EP+MCAO group compared to the MCAO group, with positive values indicating upregulation, while negative values indicate downregulation. | PMC9576414 | MI2022-2124230.008.jpg |
0.409693 | c8024044af964a49b551bb8bfec2784b | Linear discriminant analysis effect size. The biomarker of intergroup differences was found using LEfSe (n = 6). (a) The evolutionary groupings of different species are shown. Phylum to genus (or species) is depicted by a circle radiating from the center. Small circles represent different classification levels, and the diameter of the circle represents relative abundance for each level. (b) This graph illustrates the distribution of LDA values for species. In the bar chart, the colors denote groups, while the species contributions are shown by the length of the bars. | PMC9576414 | MI2022-2124230.009.jpg |
0.437892 | e0d0666caac84ee4b08c6ee39fb0041e | Selective venography of the EIVOM and LVA measurement. (A) The balloon at the proximal VOM was inflated at 7 atm, and selective venography displayed one small tortuous VOM with two distal branches, coursing upward behind the ostium of the LAA (the PentaRay catheter at the LAA). While in panel (B), after balloon inflation at 8 atm, repeated venography showed another long and straight proximal branch of VOM [invisible in panel (A)], indicating the balloon was completely occluded in panels (B) and not in panel (A). This example highlighted the importance total occlusion of the OTW before ethanol infusion. (C,D) LA substrate mapping pre- and post- EIVOM. Note the size of LVA (white arrow) markedly increased from 7.4 cm2 pre-EIVOM to 18.4 cm2 post-EIVOM. RAO, right anterior oblique; PA, postero-anterior; LSPV, left superior pulmonary vein; RSPV, right superior pulmonary vein; LIPV, left inferior pulmonary vein; RIPV, right inferior pulmonary vein; LAA, left atrial appendage; MA, mitral annulus; LVA, low voltage area. | PMC9576952 | fcvm-09-1031673-g001.jpg |
0.449431 | 012032a1e3ef41b693abfb95e4101a3f | An example of MIth linear ablation and validation of MIth block. (A) LA substrate mapping post-EIVOM displayed a patchy LVA (white arrow) in the MIth region. (B) An entire MIth lesion line was created connecting the MA and the inferior aspect of LIPV’s ostium, across the EIVOM-related LVA. Note epicardial CS ablation was applied after failure of MIth block by endocardial MIth ablation. (C,D) Tracings were surface ECG lead II, V1, PentaRay1,2 -PenraRay19,20, CS9,10 - CS1,2 and ABL1,2. (C) Pacing from PentaRay13,14 (positioned within LAA) displayed the proximal to distal CS activation sequence. The interval from LAA pacing to CS1,2 WAS 146 milliseconds (ms). (D) The ablation catheter tip (ABL) was positioned just lateral to the MIth line [shown in panel (B)]. The interval from pacing from ABL1,2 to CS1,2 was 176 ms, which was longer than that from pacing from LAA to CS1,2 (146 ms), indicating the MIth line was blocked. LSPV, left superior pulmonary vein; RSPV, right superior pulmonary vein; LIPV, left inferior pulmonary vein; RIPV, right inferior pulmonary vein; LVA, low voltage area; MIth, mitral isthmus; CS, coronary sinus. | PMC9576952 | fcvm-09-1031673-g002.jpg |
0.471121 | 47edb28e7d934ae09f450e03b1560c33 | Comparison of RF ablation parameters between the “EIVOM first” and “RFCA first” group. The average ablation index (AI) (A), contact force (B), and number of lesions (C) was significantly greater in “RFCA first” group than those in “EIVOM first” group. | PMC9576952 | fcvm-09-1031673-g003.jpg |
0.38454 | 425b009a609640539ae4e1c89f7af69b | Correlation of ethanol volume with the size of ΔLVA at the MIth region. Spearman’s coefficient = 0.66, P < 0.001. | PMC9576952 | fcvm-09-1031673-g004.jpg |
0.440018 | 955202691cc14f8087d225cb1da1133f | The ROC curve for the ethanol volume and comparison of ATa-free survival. Panel (A) displayed the ROC curve for the ethanol volume. Area under the curve (AUC) was 0.827, 95% CI: 0.686–0.967, P = 0.008. Panel (B) showed the Kaplan–Meier plots of ATa-free survival in patients with MIth block and those without during one year’s follow-up. The ATa-free survival was significantly higher in patients with successful MIth block than that in those without (Log-rank test, P = 0.01). ATa, atrial tachyarrhythmias. | PMC9576952 | fcvm-09-1031673-g005.jpg |
0.469044 | 76437ad71cef4db998a92cf93e08d215 | Central illustration: Technical aspects of EIVOM on acute MIth block. | PMC9576952 | fcvm-09-1031673-g006.jpg |
0.467282 | e7823fb14c6046d2a4bbb1fec95dde6a | Patient allocation of clinical trial. | PMC9577569 | jnm-28-4-693-f1.jpg |
0.426012 | f11e8d50ea674cbfa0dec7604eb5424b | Reduction of colonic motor activity and intraluminal high-amplitude pressure by cold saline in rats. (A) The colonic motility and intraluminal high-amplitude pressure after administration of room temperature saline. (B) A typical pattern of reduction of colonic motility and intraluminal high-amplitude pressure induced by 5°C saline. (C) Serial photographs of colonic motor activity. Left: motility before treatment. Right: motility after treatment with 5°C saline. | PMC9577569 | jnm-28-4-693-f2.jpg |
0.395295 | b5d2910f0336494bae46f1fd7f50c37c | In clinical trial, the ratio of patients who did not show any peristalsis (grade 0) after mildly cool water or room temperature water was administered as a direct spray to the colonic mucosa. *P < 0.05 in comparison to room temperature water (chi-squared test). | PMC9577569 | jnm-28-4-693-f3.jpg |
0.409861 | 85ba01a4b65d4620a0abf6e6866d968d | Suppression of peak frequency (PF) after the administration of 5°C saline in rats. (A) Decreased ratio of PF in high-amplitude pressure following the administration of 5°C saline. To quantify the decrease in high-amplitude pressure induced by 5°C saline, the ratio of contraction frequency before and after administration with a length of more than 8 mm was calculated as the %PF. *P < 0.05 vs room temperature. (B) The area under the curve (AUC) at high-amplitude pressure with 5°C saline. The AUC was calculated before and after drug administration using the lowest intraluminal pressure value as the baseline. There were no significant differences among the 3 groups. (C) The results of the peak pressure amplitude (PPA) at high-amplitude pressure with 5°C saline. The difference between PPA and the lowest intraluminal pressure was defined as PPA, and the ratio of PPA before and after medication was calculated as % PPA. There was no significant difference among the 3 groups (n = 5). | PMC9577569 | jnm-28-4-693-f4.jpg |
0.542912 | 5a755ac8e1104bca89c0edb0520654af | Colonic motility in wild type (WT) mice and transient receptor potential melastatin 8 (TRPM8)-deficient (TRPM8–/–) mice. The intraluminal high-amplitude pressure and colonic motor activity before and after administration of 5°C saline in wild type (WT) mice. A typical pattern of reduction of colonic motor activity and intraluminal high-amplitude pressure induced by 5°C saline. (D) The colonic motility and intraluminal high-amplitude pressure before and after administration of 5°C saline in transient receptor potential melastatin 8-deficient (TRPM8)–/– mice. (E) Decreased ratio of peak frequency (PF) in high-amplitude pressure following administration of 5°C saline in WT mice. To quantify the decrease in high-amplitude pressure induced by 5°C saline, the ratio of contraction frequency before and after administration with a length of more than 8 mm was calculated as the %PF. *P < 0.05 vs room temperature (25°C) of wild type mice. (F) The area under the curve (AUC) at high-amplitude pressure with 5°C saline. The AUC was calculated before and after saline administration using the lowest intraluminal pressure value as the baseline. There were no significant differences between the 2 groups. (G) The results of the peak pressure amplitude (PPA) at high-amplitude pressure with 5°C saline. The difference between PPA and the lowest intraluminal pressure was defined as PPA, and the ratio of PPA before and after medication was calculated as %PPA. There were no significant differences between the 2 groups (n = 6). | PMC9577569 | jnm-28-4-693-f5.jpg |
0.448102 | fa3769bbed4540c7ad31fbd71a37ca07 | Colonic motility in rats without or with the administration of a transient receptor potential ankyrin 1 (TRPA1) inhibitor. (A) Decreased ratio of peak frequency (PF) in high-amplitude pressure following the administration of 5°C saline in rats without administration of a TRPA1 inhibitor. To quantify the decrease in high-amplitude pressure induced by 5°C saline, the ratio of contraction frequency before and after administration with a length of more than 8 mm was calculated as the %PF. **P < 0.01 vs room temperature (25°C) without a TRPA1 inhibitor, ***P < 0.001 vs room temperature (25°C) with a TRPA1 inhibitor. (B) The area under the curve (AUC) at high-amplitude pressure with 5°C saline. The AUC was calculated before and after administration of saline using the lowest intraluminal pressure value as the baseline. There were no significant differences between the 2 groups. (C) Results of the peak pressure amplitude (PPA) at high-amplitude pressure with 5°C saline. The difference between the PPA and the lowest intraluminal pressure was defined as the PPA, and the ratio of the PPAs before and after medication was calculated as the %PPA. There were no significant differences between the 2 groups (n = 3). | PMC9577569 | jnm-28-4-693-f6.jpg |
0.441288 | bf2e1b1486d34082a4c4491ac3f19c4c | Transient receptor potential melastatin 8 (TRPM8) localization in the TRPM8 green fluorescent protein (GFP) mouse colon. (A) GFP-targeted polyclonal antibody staining in the TRPM8 GFP mouse colon. (B) GFP staining in C57BL/6J mice. No detectable signal was observed. (C) Enlarged (×2 zoom) view of GFP staining in c57bl6 mice. No detectable signal was observed. (D) Enlarged (×2 zoom) view of a section of the TRPM8 GFP mouse colon. (E) Enlarged (×2 zoom) view of a section of the TRPM8 GFP mouse colon. (E) Calcitonin gene-related peptide (CGRP) staining in the TRPM8 GFP mouse colon. (G) Merged image showing TRPM8 and CGRP expressing cells in the TRPM8 GFP mouse colon. | PMC9577569 | jnm-28-4-693-f7.jpg |
0.48353 | 30cb6aa343d749b98ee137eb4c3a521b | Flow chart of study cohort. IVF, in vitro fertilisation. | PMC9577921 | bmjopen-2022-063981f01.jpg |
0.424127 | 679163d88f724f86a08824180938e461 | Trends in prescriptions and costs of OACs over 11 years (2010 to 2020). Linear regression analysis with year as the independent variable and number of treatment visits or costs as the dependent variable, using the data from 2010 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig1.jpg |
0.402839 | c06a046021f6419292a169ab4518ecf6 | The number of treatment visits and the percentage (A) and the costs (B) for each OAC. Linear regression analysis with year as the independent variable and number of treatment visits or costs as the dependent variable, using the data from 2016 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig2.jpg |
0.438918 | c845a75846cc4bd18fe58dafdd447a1d | The number of treatment visits and the percentage for each OAC in AF (A) and VTE (B). Linear regression analysis with year as the independent variable and number of treatment visits as the dependent variable, using the data from 2016 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig3.jpg |
0.399518 | 4e87fa428113403186d5310bf3c8094d | The number of treatment visits for warfarin (A), rivaroxaban (B), and dabigatran (C) in different indications. Linear regression analysis with year as the independent variable and number of treatment visits as the dependent variable, using the data from 2016 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig4.jpg |
0.431946 | 1648c1922e1d4e8f938728070a8134c5 | The number of treatment visits for each OAC in different age groups. Linear regression analysis with year as the independent variable and number of treatment visits as the dependent variable, using the data from 2016 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig5.jpg |
0.469011 | 49fc522e0d914ff58f09b5fdd05e6b67 | The number of treatment visits for each OAC in different grades of hospitals. Linear regression analysis with year as the independent variable and number of treatment visits as the dependent variable, using the data from 2016 to 2020. | PMC9578173 | 10.1177_10760296221132551-fig6.jpg |
0.413367 | ac1a4cde486341cfa8ec7bd0b6916f9f | The research grouping flow chat | PMC9578274 | 12884_2022_5088_Fig1_HTML.jpg |
0.441482 | 54462349efa74e72871e19edc5f54453 | The comparison of fertilization outcomes between Group IVF/H and Group IVF/D. The values of the FER, CLR and HER in subgroups H/ET and H/FET are shown in the separated blue histogram and those of the D/ET and D/FET subgroups are shown in the red histogram. Note: FER = fertilization rate; CLR = cleavage rate; HER = high-quality embryo rate; * p < 0.05 **p < 0.01 ***p < 0.001 | PMC9578274 | 12884_2022_5088_Fig2_HTML.jpg |
0.448565 | 11904c4f00584899b61a7d2c295d97ba | The comparison of embryo transfer outcomes between Group IVF/H and Group IVF/D. The proportions of the BPR, CPR, LBR, MUR, MSR, TBSR, LBW and BDR in the H/ET and H/FET subgroups are shown in the combined blue histogram and those of the D/ET or D/FET subgroups are shown in the combined red histogram. Note: BPR = biochemical pregnancy rate; CPR = clinical pregnancy rate; LBR = live birth rate; MUR = multipregnancy rate; MSR = miscarriage rate; TBSR = total baby sex ratio; LBW = low birth weight rate; BDR = baby with birth defect rate;* p < 0.05 **p < 0.01 ***p < 0.001 | PMC9578274 | 12884_2022_5088_Fig3_HTML.jpg |
0.442908 | ee4bc89a86cd446db3cf3844017649a3 | Integrated on-site screening
system for COVID-19. (a) Schematic
depicting the structure and essential components of the integrated
on-site screening system for COVID-19. (b) Workflow of COVID-19 sample
screening using the integrated on-site screening system. Details are
presented in Materials and Methods section.
(c) Design of air pumping and disinfection module. (d) Design of flexible
disinfection film. (e) User interface of online FT-IR detection system
for COVID-19 sera. (f) Structure of CNN. | PMC9578365 | ac2c02337_0002.jpg |
0.451787 | 6be39ad72ef9403f9b2c4aabb15d5d1c | ANOVA results for the average spectra
of each group. (a) p values of in-pair average spectra
at each wavenumber.
The darker blue grids represent the lower p values
at the corresponding wavenumbers. Spectral differences were statistically
significant when p < 0.0001. (b) Direct comparison
in pairs of the three average in-group SD spectra. The visible differences
shown here are consistent with the p values above. | PMC9578365 | ac2c02337_0003.jpg |
0.432705 | f0aa1e2389e94108b9004ea2b274ee40 | Evaluation results of classification models.
(a) ROC of COVID-19
samples obtained from PLS-2C. (b) ROC of COVID-19 samples obtained
from CNN-2C. (c) Regression vector of COVID-19 from PLS-2C with respect
to the spectral region of 1700–900 cm–1.
VIP values reflect the importance of specific spectral regions. (d)
ROCs of three groups of samples obtained from PLS-3C. (e) ROCs of
three groups of samples obtained from CNN-3C. (f) Regression vector
of COVID-19 from CNN-2C with respect to the spectral region of 1700–1480
cm–1. | PMC9578365 | ac2c02337_0004.jpg |
0.432902 | cd350ec05fb549b2b48b1d92e5b9ee55 | Statistical results of field blind test
at Shanghai customs. (a)
Model output of PLS-2C. The direct output represents the probability
of being positive given by the classifier. (b) Model output of CNN-2C.
(c) Confusion matrix of classification results obtained from PLS-2C.
(d) Confusion matrix of classification results obtained from CNN-2C.
(e) Confusion ball visualization of sensitivity (true positive) and
specificity (true negative) obtained from PLS-2C. (f) Confusion ball
visualization of sensitivity (true positive) and specificity (true
negative) obtained from CNN-2C. | PMC9578365 | ac2c02337_0005.jpg |
0.464873 | d79c216bd7014279bd14bf60b41792a6 | Stability of manifest refraction spherical equivalent over time in the SMILE group (y-axis: spherical equivalent in diopters; x-axis: postoperative time interval in months). | PMC9578865 | JOPH2022-2625517.001.jpg |
0.434364 | 1bbe8e97d208483489a6937252a51844 | Stability of manifest refraction spherical equivalent over time in SMILE Xtra group (y-axis: spherical equivalent in diopters; x-axis: postoperative time interval in months). | PMC9578865 | JOPH2022-2625517.002.jpg |
0.424142 | 388a5c1d6f06422fbb6d1d5445887d60 | Cumulative Snellen visual acuity in SMILE group: preop CDVA and post UDVA. | PMC9578865 | JOPH2022-2625517.003.jpg |
0.439248 | 5906c6c2f21948928fd89af04495f5a4 | Cumulative Snellen visual acuity in SMILE Xtra group: preop CDVA and post UDVA. | PMC9578865 | JOPH2022-2625517.004.jpg |
0.470639 | 249ee30d305e4dfea132082074ba4890 | Thinnest point pachymetry following SMILE and SMILE Xtra. | PMC9578865 | JOPH2022-2625517.005.jpg |
0.453515 | e62624de6aa3481c8d67a3e1d9e5f2f3 | MRI (3.0T) at the time of presentation. Axial T2 Weighted Image (A), Axial Fluid attenuated inversion recovery (FLAIR) sequence (B) and coronal FLAIR (C) show cortical thickening and hyper intense signal in the medial aspect of bilateral temporal lobes Right > Left. | PMC9579373 | fneur-13-1017086-g0001.jpg |
0.374139 | a2dc687a68c340efa3709fc82adb32e0 | Results of cerebrospinal fluid using tissue-based assays (TBA). Hippocampus (A), striatum (B), cerebral cortex (C), and cerebellum (D). | PMC9579373 | fneur-13-1017086-g0002.jpg |
0.477602 | 09d1c8b19fd64f1184f921188879d4ca | Regional characteristics of the anterior epiblast in the st. 5 chicken embryo. (A) Superimposition of the region with N2 enhancer activity, Sox2 expression domain, and the map from Fernández-Garre et al. (2002) of the precursors for the brain portions, the forebrain (FB), midbrain, (MB), and hindbrain (HB). (a) The N2 enhancer activity region is shown in pale white with the outer lining shown as a magenta dashed line. (b)
Sox2 expression domain at mid st. 5 is shown by the in situ hybridization signal (purple). (c) The map by Fernández-Garre et al. (2002) drawn using homotopic grafting of labeled epiblast disks of ∼100 µm diameter is shown as a reference. The map was drawn for st. 4 embryos. However, the same map holds for early st. 5, as anterior epiblast cell migration is minimal until early st. 5. The brain precursor map was revised as shown in Figure 3 using the trajectory analysis of single-cell resolution in our study (Yoshihi et al., 2022). The anterior side is directed upward. N, the node. Scale bar, 500 µm. This panel was adapted from Yoshihi et al. (2022)
Supplementary Figure S1. (B) A translucent image of a cultured chicken embryo of the stage analogous to (A), taken from a frame of Supplementary Movie S1. A cloudy region outlined by the broken line indicates the population of epiblast cells converging to the midline. | PMC9581324 | fcell-10-1019845-g001.jpg |
0.425771 | 7fd467ce153e49c2abea0190023307e7 | Long–range axial convergence of sparsely EGFP-labeled anterior epiblast cells. (A) Snapshots of an EGFP–labeled chicken embryo at different developmental stages; excerpt frames taken from Supplementary Movie S2. (B) Trajectories of EGFP-labeled cells were drawn in random colors to distinguish individual lines during st. 5–8 of the same embryo, taken from Supplementary Movie S3. Broken ovals indicate the cell trajectories showing cell migration across the area pellucida/area opaca boundary. Scale bars, 1 mm. Adopted from Figure 1A of Yoshihi et al. (2022). | PMC9581324 | fcell-10-1019845-g002.jpg |
0.407377 | 9f9d2b329fbb4b21907c4eadc5663d2a | The distribution of head tissue precursors at st. 5. (A) Positions of the precursors for different brain portions, FB, MB, and HB, indicated by color-coded dots, compiled from data of four embryos. The data are compared with the brain portion precursor map by Fernández-Garre et al. (2002), shown as shaded areas. (B) The distribution of the precursors for the dorsal brain/head ectoderm at st. 8/9 in three embryos, color-coded according to the abutting brain portions. (C) Combination of the data in (A,B), where the larger dots represent brain portion precursors. Approximate boundaries of the above precursor regions and the outer limit of Sox2 N2 enhancer activity (Figure 1A) are drawn in cyan. N, the node position. Scale bar, 500 µm. Adopted from Figure 4 of Yoshihi et al. (2022). | PMC9581324 | fcell-10-1019845-g003.jpg |
0.391285 | 9d8c12eea9c945c48eacb4b742836b63 | The developmental outcome of the node graft labeled with mCherry depended on the graft sites. (A) Development of the node grafts from mCherry-expressing st. 4 Japanese quail embryos at various positions of the chicken host embryos: (a) Replacing the host node; (b) Anterior to the host node; (c) Posterior to the host node. Inverted U, host node; horizontal solid lines, the boundary of anterior and posterior embryonic regions; broken horizontal lines, the anteroposterior level of the node grafting; vertical dotted lines, the host embryo axes; scale bar, 500 μm; AME, anterior mesendoderm; PP, prechordal plate; ANC, anterior notochord; PNC, posterior notochord. (B) Embryos analogous to (b,c) were immunostained for SOX2. Node grafting at an anterior position always resulted in secondary brain development expressing SOX2. Scale bar, 500 µm. Adapted from Supplementary Figure S7 of Yoshihi et al. (2022). | PMC9581324 | fcell-10-1019845-g004.jpg |
0.404933 | 828f8d53e0b2410d83494a428e17be19 | AMEs extended from the grafted st. 4 node or isolated st. 5 AME elicits the gathering of surrounding epiblast cells, which develop into secondary brain tissues, as indicated by live imaging of the fluorescent-labeled epiblast and node/AME. Adopted from (B,D) of Yoshihi et al. (2022). The anterior side is at the top. Open arrowheads indicate the node/AME grafts (magenta); asterisks indicate the host nodes. Broken lines encircle the epiblast cells gathering around the gAME and forming secondary brain tissues. The inverted U-shapes in dotted lines indicate the primary neural plate regions at the prospective and forming stages. (A) Grafting an mCherry-expressing quail node at a lateral position of the EGFP-labeled host epiblast. Excerpts from Supplementary Movie S5. (B) Grafting a st. 5 quail AME isolated according to Qiu et al. (1998). Excerpts from Supplementary Movie S6. The periods in the culture are indicated in panels. (a) The stage when the quail node/AME was grafted. (b) The graft-derived AME (gAME) elongated and elicited the gathering of nearby epiblast cells (encircled by broken lines). (c) The gAME tissue differentiated into the PP (indicated by an arrow) and ANC (covered by EGFP fluorescence). The head precursors that converged on gAME started to form secondary brain tissues (encircled by broken lines). (d) The secondary brain (encircled by broken lines) fused to the primary brain at the posterior end in these specimens. Arrowheads in (b,c) indicate the contribution of cells derived from area opaca to the secondary brain tissue. Scale bar, 500 µm. | PMC9581324 | fcell-10-1019845-g005.jpg |
0.43578 | 66e2c1e1841145b88629881d7e6376ca | The brain portions that developed in the secondary brain depended on the AME graft positions. (A) Representative AME grafts at different AP levels in host embryos immediately after grafting (upper) and after ∼18 h with hybridization for Otx2 and Gbx2 (lower). The AME extended anteriorly from the graft site to a length of ∼500 µM. (a) An example of an anterior AME graft resulting in FB and MB development in the secondary brain. (b) The AME graft at the node level resulted in MB and HB portions in the secondary brain. (c) Posterior AME grafting elicited the development of inferior MB and large HB portions. In all cases, the posterior end of the secondary brain fused to the host brain at the level of the same portion, supporting the model that host and secondary brain portions develop using the pool of anterior epiblast cells of the same brain portion specificity. The horizontal bars in the upper panels extend 1 mm from the node center. Scale bar for the lower panels, 1 mm. Modified from Figure 6 of Yoshihi et al. (2022). (B) Upper: Divisions of the anterior epiblast field with distinct brain portion specificities, drawn according to the data in Figure 3C, and the schematic representation of the extended AME grafts gathering the proximal epiblast cells (arrows) from different divisions. Lower: The resultant composition of the brain portions in the secondary brain. These relationships were confirmed using 13 AME-grafted embryos. Two additional FB-only examples are shown in Figure 7. The host brain on the midline develops with all three FB, MB, and HB portions, because the midline AME passes through all three epiblast divisions. Scale bar, 500 µm. | PMC9581324 | fcell-10-1019845-g006.jpg |
0.426249 | 1058956d6f734e10ba4d9f908ff02a01 | Regionality map of the st. 4 epiblasts constructed using available data. (A) Distribution of the head tissue developmental potential of anterior epiblast cells at st. 4. The developmental potential extends by crossing the area pellucida boundary to the L5+ domain of the area opaca (Streit et al., 1997). The zones with developmental potential for the individual brain portion are drawn using pale colors. (a–c) The AME graft positions in (B). (B) Embryos with AME grafts close to the area pellucida anterior limit (arrowheads) with variable distances from the node. (C) The secondary brain tissues in embryos in (B) after 18 h, hybridized for Otx2 and Gbx2 expression and assessed for the brain portions. Scale bars, 1 mm in (A,B), 500 µm in (C). Adapted from Figure 7 of Yoshihi et al. (2022). | PMC9581324 | fcell-10-1019845-g007.jpg |
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