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Update README.md

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@@ -117,7 +117,7 @@ print(results)
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  To **find similar entities** in the text, consider the following example:
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  ```python
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- ent_prompt = "Find all "{}" mentions in the text:"
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  text = """Several studies have reported its pharmacological activities, including anti-inflammatory, antimicrobial, and antitumoral effects. The effect of E-anethole was studied in the osteosarcoma MG-63 cell line, and the antiproliferative activity was evaluated by an MTT assay. It showed a GI50 value of 60.25 μM with apoptosis induction through the mitochondrial-mediated pathway. Additionally, it induced cell cycle arrest at the G0/G1 phase, up-regulated the expression of p53, caspase-3, and caspase-9, and down-regulated Bcl-xL expression. Moreover, the antitumoral activity of anethole was assessed against oral tumor Ca9-22 cells, and the cytotoxic effects were evaluated by MTT and LDH assays. It demonstrated a LD50 value of 8 μM, and cellular proliferation was 42.7% and 5.2% at anethole concentrations of 3 μM and 30 μM, respectively. It was reported that it could selectively and in a dose-dependent manner decrease cell proliferation and induce apoptosis, as well as induce autophagy, decrease ROS production, and increase glutathione activity. The cytotoxic effect was mediated through NF-kB, MAP kinases, Wnt, caspase-3 and -9, and PARP1 pathways. Additionally, treatment with anethole inhibited cyclin D1 oncogene expression, increased cyclin-dependent kinase inhibitor p21WAF1, up-regulated p53 expression, and inhibited the EMT markers."""
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  To **find similar entities** in the text, consider the following example:
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  ```python
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+ ent_prompt = "Find all '{}' mentions in the text:"
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  text = """Several studies have reported its pharmacological activities, including anti-inflammatory, antimicrobial, and antitumoral effects. The effect of E-anethole was studied in the osteosarcoma MG-63 cell line, and the antiproliferative activity was evaluated by an MTT assay. It showed a GI50 value of 60.25 μM with apoptosis induction through the mitochondrial-mediated pathway. Additionally, it induced cell cycle arrest at the G0/G1 phase, up-regulated the expression of p53, caspase-3, and caspase-9, and down-regulated Bcl-xL expression. Moreover, the antitumoral activity of anethole was assessed against oral tumor Ca9-22 cells, and the cytotoxic effects were evaluated by MTT and LDH assays. It demonstrated a LD50 value of 8 μM, and cellular proliferation was 42.7% and 5.2% at anethole concentrations of 3 μM and 30 μM, respectively. It was reported that it could selectively and in a dose-dependent manner decrease cell proliferation and induce apoptosis, as well as induce autophagy, decrease ROS production, and increase glutathione activity. The cytotoxic effect was mediated through NF-kB, MAP kinases, Wnt, caspase-3 and -9, and PARP1 pathways. Additionally, treatment with anethole inhibited cyclin D1 oncogene expression, increased cyclin-dependent kinase inhibitor p21WAF1, up-regulated p53 expression, and inhibited the EMT markers."""
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