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as above.
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<num>. mild cardiomegaly without evidence of volume overload. <num>. left pleural effusion with associated atelectasis. underlying pneumonia cannot be excluded. <num>. left posterior eighth rib fracture of undetermined age.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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multiple bilateral pulmonary nodules and masses compatible with patient's known metastatic disease. suspected superimposed consolidation at the left lung base.
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no acute cardiopulmonary process.
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post-surgical changes in the right chest without acute chest abnormality.
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low lung volumes with bibasilar atelectasis; no radiographic evidence for acute cardiopulmonary process. stomach distended with an air-fluid level.
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<num>. standard positioning of the endotracheal tube <num>. enteric tube tip within the stomach however side port is above the gastroesophageal junction and should be advanced by approximately <num> cm for optimal positioning. <num>. emphysema.
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no consolidation. no acute intrathoracic process.
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no acute cardiopulmonary process. no evidence of free air beneath the diaphragms.
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large right basilar opacity again seen. small right pleural effusion. possible mild pulmonary edema.
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findings of diffuse chronic interstitial lung disease without evidence of superimposed acute cardiopulmonary process. a non-urgent hrct could be performed for further evaluation.
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<num>. an irregular spiculated nodule of <num> x <num> cm in the right upper lung has grown since <unk> when it was highly inconspicuous. chest ct is recommended for further evaluation. <num>. moderate cardiomegaly and enlarged bilateral pulmonary arteries suggestive of pulmonary artery hypertension. findings were discussed with dr. <unk> <unk> the significance of this nodule, and recommendation of chest ct on <unk> at <time> p.m.
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left lateral displaced rib fracture. known left-sided pneumothorax at the lung base is not clearly delineated.
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no acute cardiopulmonary process.
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stable small right pneumothorax
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no evidence of pneumonia or other abnormality. this information was telephoned to dr. <unk> at her request.
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findings suggest mild pulmonary edema.
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<num>. platelike atelectasis of left lung base <num>. small left pleural effusion.
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<num>. there is a new moderate to large left pleural effusion. <num>. right pleural effusion is similar to prior.
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subtle opacity projecting over the right upper lung which may represent prominent costochondral calcification though nodule difficult to exclude. nonemergent ct chest may be obtained to further assess.
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normal chest radiograph.
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<num>. the swan-ganz catheter tip terminates in the distal right pulmonary artery. <num>. severe cardiomegaly, unchanged compared to multiple prior studies.
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endotracheal tube is in good position. very low lung volumes. given differences in technique, the cardiomediastinal contours are stable, with widening of the superior mediastinum, could be due to fat deposition and/or adenopathy.
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no radiographic evidence of acute cardiopulmonary process.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary disease.
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<num>. no acute cardiopulmonary process. <num>. no free air below the hemidiaphragms.
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left upper to mid lung opacity, more consolidated peripherally, as also seen on recent prior pet-ct with concern for malignant involvement and/or postobstructive pneumonia.
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<num>. enteric tube terminating in the distal esophagus. this can be advanced approximately <num> cm to place the side hole beyond the gastroesophageal junction. <num>. mild pulmonary edema.
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all the monitoring devices are unchanged, persist mild pulmonary edema and moderate to large bilateral pleural effusion with atelectasis.
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no signs of pulmonary edema or other acute intrathoracic process.
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persistent pulmonary vascular prominence with interval improvement in mild interstitial edema.
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picc line terminating in the uppermost right atrium. no evidence of acute cardiopulmonary disease.
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large right pleural effusion with right middle lobe and right lower lobe collapse. the left lung is clear.
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multiple bilateral calcified pulmonary nodules and calcified bilateral hilar and mediastonal lymphadenopathy may represent sequela of patient's prior histoplasmosis infection. no defintie evidence of active cardiopulmonary process.
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mild pulmonary interstitial edema with tiny bilateral pleural effusions.
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no acute cardiopulmonary process.
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bibasilar opacities likely represent atelectasis. no focal consolidation to suggest pneumonia.
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no acute cardiopulmonary process.
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no acute intrathoracic abnormality.
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no acute cardiopulmonary process.
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no acute process. no pneumonia.
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normal chest radiograph.
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<num>. no significant interval change in numerous bilateral parenchymal opacities, consistent with metastatic disease. <num>. small bilateral pleural effusions that are new from <unk>.
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possible trace left pleural effusion. otherwise, no acute cardiopulmonary process. improved aeration of the left lower lobe.
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no acute cardiopulmonary abnormalities. no cavitary lesions
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no acute cardiopulmonary process. if concern for a rib fracture persists, dedicated rib series with markers would be recommended.
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hyperinflated lungs with coarsened lung markings compatible with known emphysema. unfolded thoracic aorta.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
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no evidence of acute cardiopulmonary process.
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hyperinflation consistent with history of copd. no radiographic evidence of pneumonia.
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no pneumonia, edema, or effusion.
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no focal consolidation. minimal pulmonary vascular congestion stable to possibly minimally increased.
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moderate bilateral pleural effusions, larger on the right than the left with associated compressive atelectasis. superimposed infection cannot be excluded.
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no acute cardiopulmonary pathology.
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<num>. no acute cardiopulmonary process. <num>. no displaced rib fracture is seen. however, if there is continued clinical concern for rib fracture, a dedicated rib series with a skin marker at the location of the patient's pain is recommended as it is more sensitive than a chest radiograph.
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no acute cardiopulmonary process. probable right basilar atelectasis.
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right subclavian picc line and left-sided pacer/aicd are unchanged in position. the heart remains stably enlarged likely reflecting cardiomegaly, although pericardial effusion should also be considered. there has been interval appearance of mild to moderate pulmonary and interstitial edema. no pneumothorax.
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no significant interval change when compared the prior study.
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<num>. no displaced fracture, however, if clinical concern for fracture persists of the ribs, suggest dedicated rib series, which is more sensitive. <num>. persistent severe enlargement of the cardiac silhouette and small bilateral pleural effusions.
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no acute cardiopulmonary process.
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<num>. probable postoperative edema of the left lung with equivocal persistence of the loculated pleural effusion underlying it. <num>. small layering pleural effusion and left basilar atelectasis. <num>. no pneumothorax.
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new retrocardiac opacity, potentially atelectasis.
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no acute cardiothoracic process.
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no acute cardiopulmonary process identified. mild stable prominence of the ascending aorta is noted, ? due to hypertension,
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no acute cardiopulmonary process. mild cardiomegaly unchanged since <unk>.
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no definite signs of pneumonia on this limited exam.
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unremarkable chest radiographic examination.
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lingular pneumonia recommendation(s): follow-up chest radiograph in <unk> weeks to document resolution.
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streaky left lower lobe opacity likely reflects minimal atelectasis.
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prominent central vessels and vascular congestion and mild cardiomegaly.
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no acute cardiopulmonary abnormality.
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no significant change.
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progressive subacute pneumonitis with possible component of pulmonary edema.
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right internal jugular pacing wire terminates in the region of the right ventricle. no large pneumothorax on this supine exam.
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as above.
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worsening bilateral peripheral opacities with a distribution suggestive of eosinophilic pneumonia.
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limited, negative.
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bibasilar opacities potentially in part due to atelectasis; however, there may be a component of infection in the proper clinical setting.
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mild cardiomegaly. no acute cardiopulmonary process.
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decreased mild pulmonary vascular congestion with decrease in left mid lung and unchanged left lower lung opacities compatible with known pneumonia. given the interval improvement, the left midlung opacity may reflect the result of an aspiration event.
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no significant interval change. no definite new focal consolidation.
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<num>. no chest radiographic evidence of pneumothorax or acute, displaced rib fracture. if localizing symptoms persist, consider dedicated rib radiograph with metallic marker placement. <num>. hyperexpanded lungs, suggesting the possibility of copd.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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stable appearance of the chest.
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<num>. there are bibasilar parenchymal opacities likely representative of chronic scarring. <num>. there is a small retrosternal nodule. follow up with ct is recommended. <num>. left pulmonary artery appears prominent suggestive of pulmonary artery hypertension or pulmonic stenosis.
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<num>. no lobar consolidation to suggest bacterial pneumonia. as describe on prior report, subtle bibasilar opacities may represent viral/atypical infection; followup imaging is recommended after therapy to document resolution. <num>. apparent right glenohumeral joint subluxation. correlate for pain.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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emphysema without a focal consolidation convincing for pneumonia.
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no acute intrathoracic process.
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<num>. no acute intrathoracic process. <num>. partially imaged left arm is notable for presence of several small metallic foreign bodies, possibly shrapnel, correlate with a prior history of gunshot or other injury.
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pulmonary edema with moderate to large bilateral effusions, larger on the right. superimposed infection particularly at the lung bases would be difficult to exclude.
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no acute cardiopulmonary abnormality.