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minimal basilar atelectasis. no displaced fracture is seen. it is difficult to evaluate the right glenohumeral joint ; correlate clinically for possible subluxation.
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<num>. no evidence of pneumonia or pulmonary edema. <num>. mild cardiomegaly, as before.
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<num>. new subtle opacity within the right upper lobe is worrisome for pneumonia. of note this is similar in location to patient's recurrent pneumonias dating back to <unk>. <num>. linear right lower lobe atelectasis.
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<num>. persistent right apical pneumothorax with improvement of right lateral basilar hydropneumothorax. <num>. increased right lower lobe opacification, which likely represents worsening atelectasis.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process.
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complex appearance at the right lung apex makes it difficult to exclude a small pneumothorax. no evidence for a large pneumothorax. right cardiophrenic opacity has is markedly improved. retrocardiac opacity is slightly more pronounced. small right effusion is new.
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since <unk>, mildly enlarging large hiatal hernia. otherwise normal chest radiograph.
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<num>. low lung volumes with bibasilar consolidations, likely atelectasis, however an underlying infectious process or aspiration cannot be completely excluded. <num>. mild pulmonary vascular congestion
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no definite acute cardiopulmonary process. increased interstitial markings persist on the left at the base which may be chronic.
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no acute cardiopulmonary abnormality.
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no evidence of acute cardiopulmonary process.
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enlargement of the ascending thoracic aorta, compatible with known history of aneurysm. no pneumonia or effusion.
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indistinctness of the hila and cephalization of vessels is consistent with pulmonary vascular congestion.
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no evidence of acute cardiopulmonary disease.
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mild interstitial edema.
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no acute cardiopulmonary process. no pneumothorax seen.
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no acute cardiopulmonary process. unchanged hyperinflation of the lungs. the aorta is tortuous, slightly more than previous studies, cannot rule out dilation of the descending aorta.
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no evidence of acute cardiopulmonary disease.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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heterogeneous right infrahilar opacity likely represents atelectasis. no strong evidence for pneumonia.
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no evidence of injury.
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no acute cardiopulmonary process, specifically, no pulmonary edema.
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<num>. evaluation is limited by low lung volumes and a dislocated right humeral head prosthesis projecting over the right upper lung. <num>. moderate cardiomegaly with moderate central pulmonary vascular congestion and mild associated interstitial edema. <num>. indistinct airspace opacities at the lung bases and right upper lung may be due to edema, atelectasis, or consolidation, depending upon the clinical scenario. <num>. indeterminate lucent lesion projecting at the level of the posterior right fourth rib may represent a benign osseous lesion, in the absence of prior studies a pulmonary parenchymal process cannot be excluded.
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no acute cardiopulmonary process. no radiographic findings to suggest active tuberculosis.
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persistent blunting of the bilateral costophrenic angles, left greater than right. evidence of extensive bilateral bronchiectasis with possible mucous plugging in the right lower lobe. stable cardiac, mediastinal, and hilar contours.
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normal chest radiographs.
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mild to moderate cardiomegaly new since <unk> without evidence of vascular congestion or interstitial edema.
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no acute cardiopulmonary process.
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<num>. right apical opacity concerning for either infection or nodule. <num>. other than low lung volumes, no significant change compared to most recent study.
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<num>. new heterogenous parenchymal opacities in the rul and rll, compatible with aspiration pneumonia. <num>. stable post-radiation changes in right paramediastinal lung.
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no pneumonia. clear lungs.
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minimal interval clearing on the right
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bibasilar opacities, likely atelectasis. an early infiltrate would be difficult to exclude. possible small left pleural effusion.
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right lower lobe pneumonia. moderate cardiomegaly.
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no change.
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no evidence of acute cardiopulmonary disease.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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increased prominence of the pulmonary vasculature is suggestive of mild fluid overload.
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no traumatic findings. if strong clinical concern for rib fracture, a dedicated rib series may be performed to further assess.
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unremarkable chest radiographic examination.
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no acute cardiopulmonary process.
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low lung volumes causing bibasilar atelectasis with superimposed right medial lung airspace opacity possibly representing aspiration.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process. no widening of the mediastinum.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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unchanged small left pleural effusion. new right lower lobe linear atelectasis. stable left lung base subsegmental atelectasis.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no evidence of acute disease.
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mild interstitial pulmonary edema. stable tiny right pleural effusion versus pleural thickening.
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no acute cardiopulmonary abnormality. no displaced fracture identified. if there is continued concern for a rib fracture, consider a dedicated rib series.
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normal chest x-ray.
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persistent moderate left pleural effusion with left basilar opacity likely reflective of atelectasis. increased atelectasis within the right lung base.
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<num>. central perihilar opacities and reticular markings diffusely overlying both lungs, which likely represent pulmonary edema. <num>. opacity overlying the left lung measuring up to <num> cm likely represents lung mass previously seen on chest ct <unk>. <num>. bibasilar atelectasis.
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pulmonary vascular congestion without frank edema or effusion.
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no acute cardiopulmonary process.
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improved aeration of the lungs with persistent mild bibasilar atelectasis.
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<num>. interval removal of left chest tube. small slightly increased left apical pneumothorax. <num>. left lateral chest subcutaneous emphysema, unchanged. <num>. minimal pneumoperitoneum noted under the left hemidiaphragm. <num>. small right pleural effusion versus pleural thickening. <num>. well-circumscribed gas collection of the left lower lateral chest wall may represent gut herniation as previously discussed.
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enteric tube coiled in the oropharynx with the tip at the thoracic inlet.
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no evidence of acute disease.
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no acute cardiopulmonary abnormality.
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no evidence of acute cardiopulmonary process. compression fractures of lower thoracic vertebrae are unchanged.
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interval improvement in bilateral pleural effusions with continued small bilateral pleural effusions.
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no acute thoracic injury.
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no radiographic evidence of pneumonia. however, if clinical suspicion persists, a chest ct may be performed for further evaluation.
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no acute cardiopulmonary process.
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possible tiny right pleural effusion, otherwise, no acute cardiopulmonary process.
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<num>. the dobbhoff tube projects over the left hemidiaphragm and should be advanced approximately <num> cm to place it safely in the stomach. <num>. persistent retrocardiac consolidation with worsening opacities at the right base and left upper lobe may reflect atelectasis, however aspiration and pneumonia in the right clinical setting cannot be excluded.
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<num>. no acute cardiopulmonary process. no radiographic evidence of etiology of patient's chest pain or cough.
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no acute cardiopulmonary process.
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bronchovascular crowding and atelectasis. no acute cardiopulmonary process.
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dobhoff tube in the stomach
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no evidence of acute cardiopulmonary process.
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<num>. no acute cardiopulmonary process. <num>. rounded dense structure overlying the right lung base is similar to prior and may represent costochondral calcification.
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<num>. the tip of the new right ij central line is seen terminating in the right atrium. recommend retraction for optimal positioning. <num>. otherwise, stable appearance of cardiomegaly, mild pulmonary edema, and dense bibasilar opacities, likely due to a combination of atelectasis and small pleural effusions, stable since prior exam.
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<num>. no focal consolidation. mild emphysema <num>. moderate hiatal hernia.
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slight interval worsening of the pre-existing parenchymal opacities compared to the prior exam from <unk>. lines and tubes are in appropriate position.
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no pneumonia, pleural effusion or pulmonary edema.
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<num>. right internal jugular central venous line terminates in the right atrium. recommend pullback by <num> cm and repeat chest radiograph. <num>. worsening right upper lobe opacity likely reflects atelectasis.
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increased bilateral atelectasis, left greater than right. no focal consolidation.
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cardiomegaly.
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findings suggestive of decompensated congestive heart failure, with pulmonary vascular engorgement, mild central pulmonary edema and bilateral pleural effusions.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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<num>. no evidence for pneumonia. there is moderate cardiomegaly, unchanged. <num>. interval placement of left ij approach hemodialysis catheter, the tip of which is projecting over the right atrium.
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persistent opacity in the right middle lobe, improved from the prior examination consistent with resolving pneumonia
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right ij has been placed with tip ending in right atrium, there is no pneumothorax. bibasilar atelectasis, more extensive in the left lower lobe increased interstitial pulmonary edema
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no focal consolidation. slight blunting of the posterior left costophrenic angle, new since <unk>, could be due to trace pleural effusion.
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no evidence of active or latent tb infection. no acute cardiopulmonary process.
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large right pleural effusion with associated compressive atelectasis in the mid-to-lower lung. findings are increased from prior exam. small left effusion and basal atelectasis is also seen.
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no evidence for pulmonary edema. postsurgical changes in the right lung lower lung field, and atelectasis in the lung bases.
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left base opacity could be due to aspiration or infection.
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no acute cardiopulmonary process.