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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17731156/s51667676/fe980453-23ed1479-a7e346b2-f9184c20-44c25643.jpg
no intrathoracic lymphadenopathy based on chest x-ray.
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no acute intrathoracic process.
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a right-sided picc terminates in the upper svc. no acute cardiopulmonary abnormality.
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left-sided pleural effusion and pulmonary opacity is unchanged. minimal right basal atelectasis. no pneumothorax.
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new left lower lobe opacification and small left pleural effusion concerning for pneumonia.
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status post endotracheal and endogastric tube removal without evidence of complication or other change.
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no acute intrathoracic process.
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stable small left pneumothorax
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ill-defined focal opacity within the right mid lung field. this could reflect an area of atelectasis but infection or inflammation cannot be excluded.
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right basilar opacity compatible with known malignancy and right lower lobe collapse with small right pleural effusion. right hilar lymphadenopathy. left lung is clear.
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slight interval improvement in left base heterogeneous opacities, follow up radiographs in <num> weeks are recommended to document complete resolution.
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no evidence of pneumonia.
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<num>. post treatment contour of involuted left mediastinal mass is stable compared to <unk>. <num>. progressive maturation of radiation fibrosis in the left lung perihilar region. <num>. no recurrence of intrathoracic malignancy.
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small nodular opacity in left upper lobe could potentially be due to an early focus of pneumonia, but lung cancer is an additional consideration. short-term followup radiograph after antibiotic therapy may be helpful to assess for resolution. alternatively, chest ct could be considered for further characterization, particularly the patient has risk factors for lung cancer moderate cardiomegaly.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18078466/s53917613/5dcab023-276046cf-b4dcc844-984ee968-c28008a5.jpg
bilateral pleural effusions and compressive lower lobe atelectasis, without significant change from ct performed earlier today.
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no acute cardiothoracic process.
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subtle opacity in the right posterior lung base could represent pneumonia in the right clinical setting.
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new moderate pulmonary edema.
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findings suggesting minor left basilar atelectasis; otherwise unremarkable.
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lobulated contour to the right medial hemidiaphragm most likely represents eventration when compared to the prior study, although diaphragmatic hernia could also have this appearance. there is likely a small hiatal hernia. lungs are well inflated without evidence of focal airspace consolidation to suggest pneumonia. right paratracheal nodular opacity likely represents a dilated vein related to a fluid replete state. no pleural effusions, pulmonary edema or pneumothorax. heart is upper limits of normal in size.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18023726/s56389584/8ec7d67e-69944753-3b40f3cd-7ef56b82-a6956483.jpg
no acute cardiopulmonary process.
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subtle haziness at the lateral left lung base which could relate to atelectasis but early consolidation is not excluded in the appropriate clinical setting.
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no interval change since the prior study. no evidence of pneumonia or effusion.
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no acute cardiopulmonary process.
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no focal consolidation. moderate cardiomegaly.
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normal chest radiographs.
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improved, but moderate pulmonary edema and unchanged, small, bilateral pleural effusions with substantial bibasilar atelectasis.
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no radiographic evidence of acute cardiopulmonary process such as pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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ng tube with tip past the ge junction with side port likely in the distal esophagus and should be advanced. new right basilar opacity suspicious for infection or potentially aspiration given history.
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new obscuration of the left heart border, possibly atelectasis, but may reflect pneumonia in the correct clinical setting.
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slight improvement in fluid overload.
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<num>. left basal opacity, likely atelectasis, cannot exclude pneumonia. <num>. apparent increase in soft tissue in the left axilla.
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relatively unchanged exam with continued diffuse parenchymal opacities and mediastinal lymphadenopathy compatible with sarcoidosis.
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cardiomegaly without superimposed acute cardiopulmonary process.
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hazy densities at the lung bases. followup to resolution is recommended.
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cardiomegaly with evidence of mild pulmonary edema, suggestive of heart failure.
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persistent right-sided pneumonic infiltrates, extension of infiltrates into left lower lobe area of moderate size.
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left basilar atelectasis. no acute cardiopulmonary process.
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stable mild pulmonary edema and moderate cardiomegaly. bibasilar opacities may represent atelectasis or infection in the appropriate clinical setting.
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increasing asymmetric pulmonary edema, right greater than left.
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no acute intrathoracic process.
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small left pleural effusion vs pleural thickening. no fracture seen.
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<num>. et tube in place in the lower trachea. <num>. lung volumes are extremely low. the heart and pulmonary arteries appear significantly enlarged, raising suspicion for a cardiac etiology.
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no acute intrathoracic process.
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mild pulmonary vascular congestion with mild bibasilar atelectasis and small bilateral pleural effusions.
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stable deformity along the right lateral rib cage. no acute findings.
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<num>. stable moderate right pleural effusion despite the presence of a right-sided drainage catheter since <unk>, suggesting possible loculation of the pleural fluid. <num>. stable bilateral lung nodules since <unk>.
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findings suggest mild vascular congestion.
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improved appearance of the lungs bilaterally with small residual right pleural effusion.
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no acute cardiopulmonary abnormality. posterior elevation of the left hemidiaphragm, of unknown chronicity.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process. no pleural effusions.
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peribronchial cuffing may reflect bronchitis or atypical edema.
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successful placement of second chest tube eliminating previously described tension pneumothorax. no new pulmonary abnormalities are identified. suggestion of right middle lobe atelectasis and some pleural effusion as before. also, the multiple left-sided rib fractures are grossly unaltered.
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<num>. bilateral pleural effusions, left greater than right. <num>. a widened paratracheal stripe suggests possible lymphadenopathy. ct is recommended for further evaluation. these findings were entered onto the critical communications dashboard by dr. <unk> at <unk> on <unk>.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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persistently increased interstitial markings in the lungs bilaterally potentially due to interstitial edema or chronic underlying lung disease. superimposed new consolidation in the left lung worrisome for superimposed infectious process. recommend repeat after treatment.
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left hilar prominence, if the suspicion is present then ct would characterize the aforementioned finding
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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persistent enlargement of the cardiac silhouette. no pulmonary edema. the lung bases are underpenetrated due to overlying soft tissue. increased opacity projecting over the inferior thoracic spine on the lateral view may be due to atelectasis although an early consolidation due to aspiration or infection is not excluded in the appropriate clinical setting.
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worsening right pneumonia and pulmonary edema.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no significant interval change. no focal consolidation to suggest pneumonia.
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unchanged mild cardiomegaly with mild pulmonary vascular congestion. minimal left basilar atelectasis without focal consolidation.
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no acute cardiopulmonary process. prominent loops of bowel in the upper abdomen, clinical correlation suggested.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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new bilateral pleural effusions.
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mild enlargement of the cardiac silhouette. central pulmonary vascular engorgement without overt pulmonary edema.
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no evidence of acute cardiopulmonary disease.
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parenchymal opacities throughout the left lung and right upper lung which could be infection in the proper clinical setting. increased pleural based opacity on the right, potentially pleural-based thickening and/or possible effusion. consider pa and lateral to further assess these findings. followup will certainly be necessary.
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no acute cardiopulmonary abnormality.
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normal chest x-ray.
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interval progression of right lower lobe opacification, with associated pleural thickening, effusion and volume loss. recommend chest ct examination for further evaluation. findings were conveyed by dr. <unk> to dr. <unk> <unk> telephone at <time>pm on <unk>, <num> minutes after discovery.
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<num>. interval improvement in previously noted mild pulmonary edema since <unk>. <num>. persistent small right apical pneumothorax. <num>. unchanged bilateral small pleural effusions and bibasilar atelectasis since <unk>. <num>. an air-fluid level projects over the right mainstem bronchus, likely representing the neo esophagus.
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lingular pneumonia.
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low lung volumes and bibasilar atelectasis.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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stable examination from <unk>.
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no acute findings in the chest.
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<num>. stable small left apical pneumothorax. <num>. interval improvement in the subdiaphragmatic free air compared to the prior exam, with mild residual subdiaphragmatic free air. <num>. interval improvement in mild bilateral pulmonary edema.
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no acute cardiopulmonary process or evidence of osseous injury, within the limitations of a chest radiograph.
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<num>. chronic lung changes, but no evidence of pneumonia. <num>. mild vascular congestion.
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interval resolution of left pleural effusion compared with prior.
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no acute intrathoracic process.
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mild vascular congestion with mild cardiomegaly again noted.
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no evidence of pneumonia.
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<num>. bilateral pleural effusions. <num>. no evidence of pulmonary edema. no focal consolidations.
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no acute cardiopulmonary process - discussed with <unk> at <time> on <unk> by <unk> over the phone.
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bibasilar subsegmental atelectasis. otherwise, no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process. no pneumothorax.
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no acute cardiopulmonary abnormality.
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no evidence for thoracic injury. left scapula fracture.