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0.398422 | 2d84c047001344ea8ad3c2dfe99907ec | Theoretical framework for the design and implementation of a management or transmission of knowledge program. | PMC10116858 | fpsyg-14-1124650-g0002.jpg |
0.441062 | 5a60366a55e847059c4c030c91d9c0a0 | Summary of experimental results of the detection system using the ensemble of bagging trees: Performance evaluation metrics for the binary classes and the overall | PMC10117275 | 521_2023_8592_Fig10_HTML.jpg |
0.363467 | ab979c17e17d49a2829a1bad6745cefb | Phishing website example | PMC10117275 | 521_2023_8592_Fig1_HTML.jpg |
0.409489 | 07ac89a7332f4ae7aa925e3599a751e3 | Phishing URLs number reported to APAC from the first Quarter of 2019 to the first Quarter of 2022 | PMC10117275 | 521_2023_8592_Fig2_HTML.jpg |
0.445716 | 51d2e71347574863b5eea2fbf7a0e3c9 | The dataflow diagram of the proposed Malicious URL Detection and Classification System | PMC10117275 | 521_2023_8592_Fig3_HTML.jpg |
0.424046 | 114e4155dfa64bff883074ebdd6e6474 | Data preparation phases (from top-left toward bottom-right): A Data cleaning, B Feature Selection, C Dataset Shuffling, and D Dataset Division | PMC10117275 | 521_2023_8592_Fig4_HTML.jpg |
0.448505 | 90cd2032541f48bca24ed286be6cf4e7 | Summary of the performance assessment system of measurement | PMC10117275 | 521_2023_8592_Fig5_HTML.jpg |
0.434896 | c209dadd6126494cbfbcd9b385ae0692 | The typical deployment of IDS/IPS on an enterprise | PMC10117275 | 521_2023_8592_Fig6_HTML.jpg |
0.446084 | 09bb5ddc9a7d48f7ad31120146d43470 | Summary of experimental results for the performance of using Model 1(En_Bag), Model 2 (En_kNN), Model 3 (En_Bos), and Model 4 (En_Dsc) | PMC10117275 | 521_2023_8592_Fig7_HTML.jpg |
0.443174 | 5a63e45ac9364662ad65c07ccffc9e75 | Confusion Matrix of En_Bag Based System for a 2-Classes Detection Model [0 for benign and 1 for anomaly] b 5-Classes Classification Model | PMC10117275 | 521_2023_8592_Fig8_HTML.jpg |
0.436327 | 668f5a465c8b4f11825ce88f54aaeb78 | Summary of experimental results of the detection system using the ensemble of bagging trees: Performance evaluation metrics for the binary classes and the overall | PMC10117275 | 521_2023_8592_Fig9_HTML.jpg |
0.466741 | 7988dcca5bcd4740911b1dc8b5f129cc | Amplification curve in real-time RT-PCR showing positive and negative samples for pan enteroviruses. RT-PCR: Real-time polymerase chain reaction | PMC10118210 | JGID-15-13-g001.jpg |
0.466695 | 30e0ca9761fb4d26bc71384ff2a0d3ab | Genotyping shows positivity for pan enteroviruses (120 bp), HEV-71 (243 bp), CVA-6 (106 bp), and CVA-16 (225 bp). HEV: Human Enterovirus, CVA: Coxsackie virus | PMC10118210 | JGID-15-13-g002.jpg |
0.438949 | 8faf056a696d41a4b369abe4bb65de71 | Atypical presentation of HFMD. HFMD: Hand, foot, and mouth disease | PMC10118210 | JGID-15-13-g003.jpg |
0.467464 | 8bdcab931e124658b4f99d9957262dfd | ECG shows ST elevation in inferior leads with first degree heart block. | PMC10118898 | CRIC2023-9986712.001.jpg |
0.41583 | 1a924610cfc841ceb30faaaf07801681 | Cerebral CT angiography showing non-opacification of distal ICA and MCA (arrow). | PMC10118898 | CRIC2023-9986712.002.jpg |
0.491249 | 3ac9ba1d7d55448681451fe90651262a | CT cerebral angiogram shows (a) distal ICA occlusion with non-visualization of M1 MCA, (b) stentriever in situ with left ICA run showing a filling defect in distal ICA proximal M1 MCA (arrow), and (c) complete recanalization of ICA MCA. | PMC10118898 | CRIC2023-9986712.003.jpg |
0.395468 | 8f29f285b995459a820bfe6fbfe96595 | CAG showing (a) diffusely diseased RCA with maximum stenosis of 80–90% at the mid part with 100% occlusion of AM, (b) CTO of LAD from the mid part and small caliber Left circumflex artery, and (c) TIMI 3 flow after RCA revascularization. | PMC10118898 | CRIC2023-9986712.004.jpg |
0.493084 | 65858398c9174fc5ab7103f0e25f87eb | DREAM mRNA expression by quantitative PCR in benign and malignant thyroid lesions (P < 0.0001). | PMC10118984 | 2359-4292-aem-62-02-0205-gf01.jpg |
0.454631 | d9fbacaf02bf45629b302d0025608152 | DREAM mRNA expression by quantitative PCR in goiter, follicular adenoma (FA), follicular variant papillary thyroid carcinoma (FVPTC) and classic papillary thyroid carcinoma (CPTC). | PMC10118984 | 2359-4292-aem-62-02-0205-gf02.jpg |
0.513187 | 08e381017894429db0a88a4d0b43a7f7 | Kaplan-Meier curve comparing patterns of recurrence-free survival between patients according to DREAM gene expression. | PMC10118984 | 2359-4292-aem-62-02-0205-gf03.jpg |
0.374133 | 681efe96f4784393bc66728b8f1b291a | Study overview.Schematic summarising our analytic framework using iPSC-derived motor neurons (iPSMNs) and post-mortem tissue to interrogate perturbations across the spectrum of ALS. Made with BioRender. | PMC10119258 | 41467_2023_37630_Fig1_HTML.jpg |
0.422257 | 21e577b10e2f4f119d4751b43933bef7 | Differential gene expression in ALS versus control iPSMNs.a Volcano plot of differential gene expression in ALS versus control iPSMNs using the Wald test. b Functionally overrepresented terms in up-regulated (red) and down-regulated (blue) differentially expressed genes using the hypergeometric test. c GSEA of signal transduction by p53 (GO:0072331, n = 264) in ALS versus control using the permutation test. NES, normalized enrichment score. d PROGENy signalling pathway activities in ALS versus control using the weighted mean method. Pathways increased in ALS are red and pathways decreased are blue. *** represents P < 0.0001 and *P < 0.05 (p53 p < 0.001, MAPK p < 0.001, WNT p = 0.03). e Expression changes of p53 signalling pathway genes in ALS versus control according to their PROGENy weights. Genes increasing p53 activity in ALS are red whilst genes decreasing p53 activity in ALS are blue. f Activities of 429 transcription factors in DoRothEA inferred from their regulon expression changes in ALS versus control. The normalised enrichment score in ALS versus control (x-axis) is plotted according to the enrichment test p-value (y-axis). | PMC10119258 | 41467_2023_37630_Fig2_HTML.jpg |
0.42566 | bf0eed5c03074183953d9c0fd37e4ec6 | Gene expression changes in each ALS genetic background.a–e Volcano plots comparing ALS iPSMNs to controls in each ALS genetic background. Genes coloured red are significantly increased in the ALS subgroup and genes coloured blue are decreased in the ALS subgroup using the Wald test. f Heatmap showing the Pearson’s correlation coefficient for transcriptome-wide changes between each genetic background. g PROGENy p53 signalling pathway (left) and Dorothea TP53 transcription factor regulon (right) activities amongst each of the genetic backgrounds independently using the weighted mean method. **** represents P < 0.0001, ***P < 0.001, **P < 0.01, *P < 0.05. | PMC10119258 | 41467_2023_37630_Fig3_HTML.jpg |
0.415666 | 7b6bb20d88904785b4bbbace7c8e938c | Post-mortem spinal cord shows p53 activation.a Volcano plot of differential gene expression in ALS versus control post-mortem spinal cord using the Wald test. b Functionally enriched terms in up-regulated (red) and down-regulated (blue) differentially expressed genes using the hypergeometric test. c GSEA for signal transduction by p53 in ALS versus control post-mortem spinal cord using the permutation test. NES, normalized enrichment score. d PROGENy signalling pathway activities in ALS versus control post-mortem tissue using the weighted mean method. Pathways increased in ALS are red and pathways decreased are blue. e Expression changes of p53 signalling pathway genes in ALS versus control according to their PROGENy weights. Genes in ALS increasing p53 activity are red and genes decreasing p53 activity are blue. f Activities of 429 transcription factors in DoRothEA inferred from their regulon expression changes in ALS versus control post-mortem tissue using the enrichment test. g Scatterplot of ALS vs control gene expression changes in iPSMNs (x-axis) against post-mortem tissue (y-axis) using the Wald test statistic. h Heatmap showing the Pearson’s correlation coefficient for transcriptome-wide changes between each genetic background in post-mortem tissue. i PROGENy p53 signalling pathway (left) and DoRothEA TP53 transcription factor regulon activity (right) amongst each of the genetic backgrounds in post-mortem tissue using the weighted mean method. *** represents P < 0.0001 and *P < 0.05. | PMC10119258 | 41467_2023_37630_Fig4_HTML.jpg |
0.390916 | 8aa75ab0b89d40edb6cf691a0867ab3b | Alternative splicing alterations in ALS iPSMNs.a Splicing analysis of ALS and control iPSMNs with MAJIQ50. b, f Differential splicing in ALS versus control iPSMNs and post-mortem using the TNOM test. Events with P < 0.05 and Δ PSI (ALS - CTRL) > 0.1 are coloured red and < −0.1 blue. c, g Functionally enriched terms amongst genes with differential alternative splicing in iPSMNs and post-mortem using the hypergeometric test. d, h Categorisation of differential local splice variants into basic splicing types using MAJIQ modulizer in iPSMNs and post-mortem. e, i Violin plots showing PSI values (y-axis) for ALS (red) and control samples (blue) for splice events in iPSMNs and post-mortem with p-values from the TNOM test. ** represents P < 0.01 and *P < 0.05. | PMC10119258 | 41467_2023_37630_Fig5_HTML.jpg |
0.504304 | e7f2af2e381c4c2a8dc2f5b8cb6a4918 | ALS iPSMNs and post-mortem tissue accumulate somatic mutations and gene fusions.a Violin plots showing the partial residuals of somatic mutations, controlling for age and read depth, identified in Answer ALS iPSMNs in ALS (red, n = 238) and CTRL (blue, n = 42) samples, for all mutation types, insertions, deletions, and single-nucleotide variants (SNV). Statistics are from the generalised linear model Wald test using a Poisson distribution. b Forest plot showing the generalised linear model point estimate and 95% confidence interval of changes in mutation types (SNV, blue; insertion, red; deletion, green) in ALS genetic subgroups versus controls. The vertical dashed line indicates no difference, to the right of the dashed line indicates an increase in ALS. c, d As for (a, b) except in NYGC post-mortem spinal cord samples (n = 214 ALS, n = 57 controls). In addition to age and read depth, the sequencing instrument is also controlled for. e Violin plots showing the partial residuals of gene fusions in CTRL (blue, n = 90) and ALS (red, n = 306) in paired-end sequenced iPSMNs, controlling for age, read depth and dataset. Statistics are from the generalised linear model Wald test using a Poisson distribution. f Forest plot showing the generalised linear model point estimate and 95% confidence interval changes in each genetic subtype versus controls. g, h As for (e, f) except in post-mortem (n = 214 ALS, n = 57 controls), controlling for age, read depth, dataset and sequencing instrument. In the boxplots, whiskers (error bars) represent 1.5 times the interquartile range, the hinges correspond to the first and third quartiles, and the centre represents the median. **** represents P < 0.0001, ***
P < 0.001, **P < 0.01, *P < 0.05. | PMC10119258 | 41467_2023_37630_Fig6_HTML.jpg |
0.401971 | d9ca40f603e344a6871ae79d2a6ae8fe | The parity of the Holstein and the Jersey cattle.(A) Proportion of parities in the Holstein and the Jersey cattle. (B) Proportion of parities in the Holstein and the Jersey cattle exposed to heat stress (temperature-humidity index > 72). | PMC10119450 | jast-65-2-324-g1.jpg |
0.419994 | 0dc09feadb004306b88b60a031476d5d | Holstein and Jersey breeds CR considering temperature-humidity index thresholds from 50 to 78 on the day of AI.CR, conception rate; AI, artificial insemination. | PMC10119450 | jast-65-2-324-g2.jpg |
0.393609 | 29c16050e00146e3971260ad5025c6ba | Intracellular ROS levels in metaphase II oocytes.(A) Representative images of stained (green) intracellular ROS levels in the Holstein and the Jersey oocytes under NHS and HS conditions. (B) Relative intensity of ROS levels in the different breeds under NHS and HS conditions. Significantly different level: *p < 0.05, **p < 0.01, ***p < 0.001. ROS, reactive oxygen species; NHS, non-heat stress; HS, heat stress. | PMC10119450 | jast-65-2-324-g3.jpg |
0.497552 | e593a3471ced4a42ba0b72719fb2c3f6 | Intracellular mitochondrial distribution in MII oocytes.(A) Representative images of stained (MitoTracker Red) mitochondria in the Holstein and the Jersey oocytes under non-heat stress and heat stress conditions. Scale bar = 20 μm. (B) Mitochondrial distribution pattern expressed as a relative fluorescence intensity value by drawing a line across the MII oocytes. Each graph in Fig. 2 corresponded with the same position (row and column) in Fig. 2A. ZP, zona pellucida; MII, metaphase II. | PMC10119450 | jast-65-2-324-g4.jpg |
0.4192 | 2c7f4ca27c734b1ea6f9cc0b43fc6c4e | (a) The PL excitation and (b) emission spectra, and (c) photographs of g-C3N4-Ph synthesized using melamine and quinazoline-2,4(1H,3H)-dione at molar ratios 12 : 1, 14 : 1, 16 : 1, 18 : 1, 20 : 1, 22 : 1 and 24 : 1, respectively. λex = 326 nm, λem = 508 nm. | PMC10120611 | d3ra00473b-f1.jpg |
0.40311 | 22fb58e2ec3246f5bd726bcd81897e52 | (a) The emission spectra of LEDs No. a–h. Insets on the right: photographs of the LEDs in the working state. (b) The CIE coordinates of LEDs No. a–h. | PMC10120611 | d3ra00473b-f10.jpg |
0.552773 | bfeda7b1bebb4fc989801c10fd85e3fc | The solid-state 13C NMR spectra of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph. (Inset) The representative structural units of g-C3N4-Ph and s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f2.jpg |
0.550635 | 8440eb89d801443ea3dffe0aa92667e6 | The FT-IR spectra of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f3.jpg |
0.557526 | ab2ad09058b54f40bf521ba3428105c6 | Powder XRD patterns of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f4.jpg |
0.386425 | f248d7fb03234fd48755336f2298e322 | The SEM images of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f5.jpg |
0.409008 | 54615532952f4875bd652a1af7bb5d58 | (a) The UV–Vis–NIR DRS and (b) the corresponding Tauc plots of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f6.jpg |
0.463843 | 1fc81b7bbfa54e39a9e61db2aa1f91ea | (a) The PL excitation and (b) emission spectra of g-C3N4 (λex = 361 nm, λem = 467 nm), g-C3N4-Ph (λex = 326 nm, λem = 508 nm) and s-g-C3N4-Ph (λex = 463 nm, λem = 517 nm). (Inset) From bottom to top, photographs of g-C3N4, g-C3N4-Ph and s-g-C3N4-Ph under a 365 nm UV lamp. | PMC10120611 | d3ra00473b-f7.jpg |
0.53371 | 56c1f88b2eec423a83458b0e50949570 | The TG curve of s-g-C3N4-Ph. | PMC10120611 | d3ra00473b-f8.jpg |
0.489746 | 26662f414f854a88982acc7be9e55f87 | The temperature-dependent PL spectra (λex = 463 nm) of the s-g-C3N4-Ph powder measured at every 20 °C interval from 20 °C to 200 °C. (Inset) The relative PL emission intensity at the λem,max of the s-g-C3N4-Ph powder as a function of temperature. | PMC10120611 | d3ra00473b-f9.jpg |
0.488053 | 65a9bad430be48629d75709ed969457b | A hybrid approach improves reliability and reduces human effort for coding ethnographic interviews. | PMC10120879 | nihms-1839383-f0001.jpg |
0.419238 | b0463cf5a0af4b2581a43f54741a09fb | Railway transport planning process. | PMC10121017 | pone.0284747.g001.jpg |
0.403103 | 4a08df2ad5544751a8a6966b5fe99f9b | Three levels of the network in high-speed railways. | PMC10121017 | pone.0284747.g002.jpg |
0.49294 | 1246270415d1420ebc74c089ddf7066a | An illustration for timetable with multiple train types. | PMC10121017 | pone.0284747.g003.jpg |
0.428012 | 2133386f9f3743a18724eaae80aacc61 | Departure headway in different situation of train stops. | PMC10121017 | pone.0284747.g004.jpg |
0.427745 | b7ccc61752d54cdc92077120697d9d82 | Arrival headway in different situation of train stops. | PMC10121017 | pone.0284747.g005.jpg |
0.438575 | 7a65e9b4d1fa47bfbe3056698e277c6f | Beijing-shanghai high-speed railway corridor. | PMC10121017 | pone.0284747.g006.jpg |
0.424462 | ad87e254b02e49318ef8835fb669a1c0 | Optimized train stop plan of Beijing–Shanghai high-speed railway. | PMC10121017 | pone.0284747.g007.jpg |
0.421072 | 0b6ff562118741d4803e28b22f7a12f1 | Changes in the frequency of stops at each station and the connection with passenger flow. | PMC10121017 | pone.0284747.g008.jpg |
0.374057 | 43adc2f8651148c28a8d0e0e24975de7 | Time-distance diagram of optimized timetable. | PMC10121017 | pone.0284747.g009.jpg |
0.534007 | 93c8a66cef6f4528800c917f7c387188 | Contrast between train travel time and departure time deviation. | PMC10121017 | pone.0284747.g010.jpg |
0.504111 | e92a9d9e6ed0413eba18a2a996aae8bb | Mean plasma concentration-time profiles for DMT on a (A) linear and (B) logarithmic scale for all dose cohorts of DMT. DMT N,N-dimethyltryptamine | PMC10122081 | 13318_2023_822_Fig1_HTML.jpg |
0.428159 | bb1d191b67c34d4d8c43a59125f49568 | Cmax (A) and AUClast (B) for all doses of DMT. Linear regression equations: (A) Cmax = − 11.0 + 3.83·dose; (B) AUClast = − 24.2 + 42.9·dose. DMT N,N-dimethyltryptamine | PMC10122081 | 13318_2023_822_Fig2_HTML.jpg |
0.531458 | 9e690371823e4ccca464476dcfda1f96 | AnalysisComparison 1: Training for CBE versus no training, Outcome 1: Early stage (0+I+II) diagnosis | PMC10122521 | tCD012515-CMP-001.01.jpg |
0.425732 | 3dbd82b7883e4d28b3359183fabc685b | AnalysisComparison 1: Training for CBE versus no training, Outcome 2: Late stage (III+IV) diagnosis | PMC10122521 | tCD012515-CMP-001.02.jpg |
0.443185 | 58fcf405c83943f88067d2cfcc6354a4 | AnalysisComparison 1: Training for CBE versus no training, Outcome 3: Breast cancer mortality | PMC10122521 | tCD012515-CMP-001.03.jpg |
0.444983 | 5cc36b4d00614f98a63b39fb57d8e8b3 | Study flow diagram. | PMC10122521 | tCD012515-FIG-01.jpg |
0.389483 | 5900768f22bd4f3bba951609458df095 | Risk of bias summary: review authors' judgements about each risk of bias item for each included study. | PMC10122521 | tCD012515-FIG-02.jpg |
0.416968 | c327f861cd1845188aced235c8a8d2e9 | Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies. | PMC10122521 | tCD012515-FIG-03.jpg |
0.490097 | e597861064f14f7186dae493640c1ae6 | Cerebrovascular event characterization flow chart.BAV indicates bicuspid aortic valve; BAVi, cerebral or retinal infarction caused by calcific BAV thromboembolism; CVE, cerebrovascular event; ESUS, embolic stroke of undetermined source; LAi, non‐BAV, large artery atherosclerotic or lacunar cerebral or retinal infarction; nLAi, non‐BAV, non‐large artery embolic cerebral or retinal infarction; and TIA, transient ischemic attack. | PMC10122894 | JAH3-12-e028789-g001.jpg |
0.424488 | be8f8cad47224148ab23f7d2effda1d6 | Cerebral and retinal infarction in patients with bicuspid aortic valve.Of 5401 patients with BAV diagnosis, there were 83 patients with confirmed ischemic cerebral or retinal infarction. The mechanism of infarction was embolic in 72/83 (87%). Greater than one‐third of embolic infarction was diagnosed as caused by a calcific BAV, while 35% was from a non‐BAV source, and 29% was from an unknown embolic source. Patients with a calcific BAV embolism commonly had a retinal infarction. BAV indicates bicuspid aortic valve; and ESUS, embolic stroke of undetermined source. | PMC10122894 | JAH3-12-e028789-g002.jpg |
0.456746 | 888c45ffad9a4a32ad54bea6fdc94826 | Percentage of children (3–5 years) attending pre-primary education by major states. Source: Authors’ estimation from 75th NSO round data, 2017–2018 | PMC10123015 | 40723_2023_117_Fig1_HTML.jpg |
0.473908 | 637ffc261d674084872124f394df81fb | Predicted probabilities of attending pre-primary education by location and household head’s education status | PMC10123015 | 40723_2023_117_Fig2_HTML.jpg |
0.466971 | 45188923067247a8bdd8ba29354eccd3 | Predicted probabilities of attending pre-primary education by location and household consumption expenditure | PMC10123015 | 40723_2023_117_Fig3_HTML.jpg |
0.478859 | bd69c71be1d44332b289aa174e02f70c | Kaplan-Meier curves of progression-free survival based on treatment and according to TILs (panel A), Immunoscore (panel B), Immunoscore-IC (panel C) and PD-L1 expression (panel D) in the proficient mismatch repair population of the AtezoTRIBE study. PD-L1, programmed death ligand-1; TILs, tumor-infiltrating lymphocytes. | PMC10124320 | jitc-2022-006633f01.jpg |
0.432216 | bf708f0d9b4c44bcadcbd7964117a547 | Forest-plot according to TILs, Immunoscore, Immunoscore-IC and PD-L1 expression of progression-free survival in the overall population and in the pMMR subgroup of the AtezoTRIBE study. PD-L1, programmed death ligand-1; TILs, pMMR, proficient mismatch repair; TILs, tumor-infiltrating lymphocytes. | PMC10124320 | jitc-2022-006633f02.jpg |
0.421911 | 031db9b2dd91470388de18b90f768dc0 | Donut plot showing concordance among TILs, Immunoscore and Immunoscore-IC in overall population (panel A) and in proficient mismatch repair subgroup (panel B) of the AtezoTRIBE study. TILs, tumor-infiltrating lymphocytes. | PMC10124320 | jitc-2022-006633f03.jpg |
0.498515 | 920619e15ee44023abcc48c4e0fc435a | Kaplan-Meier curves of progression-free survival according to TILs (panel A), Immunoscore (panel B), Immunoscore-IC (panel C) and PD-L1 expression (panel D) in the proficient mismatch repair treated with FOLFOXIRI/bevacizumab of the AtezoTRIBE study. PD-L1, programmed death ligand-1; TILs, tumor-infiltrating lymphocytes. | PMC10124320 | jitc-2022-006633f04.jpg |
0.379212 | 34b25face1f345ddbbf83ae6f14d67d0 | The G4 DNAzyme-based colorimetric assay and CRISPR-Cas12a-mediated G4 cleavage. (A) Scheme of the color reaction from colorless to green catalyzed by the G4-Hemin complex. (B) The performance of the color reaction in the presence of different reactants. After color reactions, photographs of tubes 1–9 with different reactants were taken from the top view. (C) Monitoring the color reaction by measuring the tubes’ absorption at 420 nm with different reactants. Tubes 1–9 contained different reactants as shown in Fig. 1B. (D) The illustration of interrupting the color reaction by Cas 12a-mediated G4 cleavage. (E) The native PAGE characterization for Cas12a-mediated G4 cleavage. Cleavage time: 20 min (F) Monitoring the color reaction by measuring the tubes’ absorption at 420 nm after Cas12a-mediated G4 cleavage. G4 in tubes 1–6 were treated with different cleavage conditions as performed in the native PAGE analysis. | PMC10125216 | gr1_lrg.jpg |
0.45317 | 4845c63fc72b450e851253fabe431869 | Performance of the one-tube RT-RPA/CRISPR-Cas/G-quadruplex-based assay. (A) Illustration of the direct one-tube RT-RPA/CRISPR-Cas/G-quadruplex-based assay. (B) Performance of the direct one-tube RT-RPA/CRISPR-Cas/G-quadruplex-based assays for a different time. (C) Illustration of the glycerol-aided one-tube RT-RPA/CRISPR-Cas/G-quadruplex assay. (D) Performance of the glycerol-aided one-tube RT-RPA/CRISPR-Cas/G-quadruplex assays for 20 min. | PMC10125216 | gr2_lrg.jpg |
0.452959 | 71441b8776704a598c450057ffad7a7b | The integration of RNA extraction-free workflow with a one-tube assay for SARS-CoV-2 detection is feasible. (A) Schematic illustration of the procedures for RNA extraction. (B) The native PAGE characterization for RT-RPA products using different RNA extraction methods. (C) Performance of the combined different RNA extraction methods with the one-tube assay. (D) Cas12a-crRNA concentration-dependent effects on combined RNA extraction-free workflow with one-tube assay performance. (E) Effects of reaction time on the RNA extraction-free workflow coupled with one-tube assay. | PMC10125216 | gr3_lrg.jpg |
0.449433 | 8f62f46dac2a44cb9caaa19f4d400e04 | Temperature profile of SBC pits. Square dot line (), long dash line (), solid line () is air tempearture, soil temperature, and core temperature, respectively. | PMC10140975 | pathogens-12-00628-g005.jpg |
0.461049 | 97f7dee8133e4355bd0817e70e7a5862 | Scatter plot of Ct of post-burial spleen and femur bone marrow samples. Green dots are the Ct values of femur bone marrow samples. Orange triangles are the Ct values of spleen samples. | PMC10140975 | pathogens-12-00628-g006.jpg |
0.378032 | 0aabf695bf68423fb80c76f1c7e44b2f | Plot of log10 of the ASFv concentration for spleen samples tested with 99.8% confidence and prediction intervals. Green dots are the raw cell culture data. The green line is the best fit simple linear regression line for log10 of the ASFv concentration regressed on days since burial. The pink shaded area is the 99.8% confidence interval for the best fit regression line. The dashed blue lines represent the lower and upper bounds of the 99.8% prediction interval for the plotted regression line. The orange dot is the estimated mean number of days to log10(0) ASFv concentration, equivalent to 1 ASFv particle. The orange line or interval around the orange dot is the 99.8% prediction interval for the estimated number of days to log10(0) ASFv concentration. | PMC10140975 | pathogens-12-00628-g007.jpg |
0.434658 | 648cff3effbe4e6b8f245c215d2ddcba | Plot of days since burial vs. log10 of the ASFv concentration for femur bone marrow samples tested with 99.8% confidence and prediction intervals. Green dots are the raw cell culture data. The green line is the best fit simple linear regression line for log10 of the ASFv concentration regressed on days since burial. The pink shaded area is the 99.8% confidence interval for the best fit regression line. The dashed blue lines represent the lower and upper bounds of the 99.8% prediction interval for the plotted regression line. The orange dot is the estimated mean number of days to log10(0) ASFv concentration, equivalent to 1 ASFv particle. The orange line or interval around the orange dot is the 99.8% prediction interval for the estimated number of days to log10(0) ASFv concentration. | PMC10140975 | pathogens-12-00628-g008.jpg |
0.506772 | fc94b78bf06042c5aaba3dfec0bedbb1 | The decomposition of carcasses in SBC pits at day 1, 3, 5, 7, 14, 21, 28, 35, 56, and 144. | PMC10140975 | pathogens-12-00628-g009a.jpg |
0.511064 | 8d148636210c4a23ae5636182fee0fc8 | Expression difference of DEGs. (A) DEGs expression distribution at 4 h exposure. (B) expression distribution of DEGs at 24 h exposure. | PMC10141105 | metabolites-13-00471-g001.jpg |
0.544247 | 7cda46d29a534ecbaf9c5f6ce644cb37 | DEG distributions between two time points. Different colors represent different DEGs expression distribution. | PMC10141105 | metabolites-13-00471-g002.jpg |
0.391782 | f7a94bd2b12642ed9b6ac663fff9001a | Expression clustering of DEGs. A row indicates expressions of a DEG in each group; each column represents the expressions amount of all DEGs in a group. | PMC10141105 | metabolites-13-00471-g003.jpg |
0.473179 | 93eb3fc8314a43acaefbd09a3bbeab22 | Top 10 significant GO terms. Ordinate indicates DEG numbers; abscissa stands for specific terms. | PMC10141105 | metabolites-13-00471-g004.jpg |
0.413547 | 61b6f23c514a40ccbdd17f9302848983 | Level-2 KEGG signaling pathways statistics. | PMC10141105 | metabolites-13-00471-g005.jpg |
0.426956 | e3f46c526871464da8b8617e7428e911 | The PPI network. The dots stand for proteins, and the connection represents the interaction between genes. | PMC10141105 | metabolites-13-00471-g006.jpg |
0.46583 | abebd3232d0a48cbb7d2d65a22109ba1 | Gene expression verification (qRT-PCR). The abscissa represents Cu exposure time; the ordinate stands for fold change. a, b, and c represent significance of changes in the expression of key genes within 24 h of Cu exposure. | PMC10141105 | metabolites-13-00471-g007.jpg |
0.458424 | 73e77ef2fe044c07b65bfa4aa41c2a00 | Data acquisition of the hospitalized patients with X-linked diseases infected with Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) in Brazil during the first two years of the coronavirus disease (COVID)-19 pandemic (December 29, 2019, to March 20, 2022). We presented the total number of patients affected by severe acute respiratory syndrome and the exclusion criteria. Moreover, we presented the frequency of the diseases according to our data. We presented the data as the number of patients and their percentage. ∗, Adenovirus, Metapneumovirus, Rhinovirus, Parainfluenza (1, 2, 3, and 4), and Respiratory Syncytial Virus. | PMC10141191 | gr1_lrg.jpg |
0.4862 | 51abfe2c5c844af496031805aaeb7443 | Summary of the outcomes of the hospitalized patients with X-linked disease and infected with Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) [coronavirus disease (COVID)-2019] in Brazil included in the serial case report. | PMC10141191 | gr2_lrg.jpg |
0.446592 | 4931e23ac7334694b1f43eb295cbfb12 | Study flowchart. * Exclusions concerned 2 patients who were below 18 years old at the time of the study. | PMC10142708 | medicina-59-00722-g001.jpg |
0.450541 | 2d0393a6cf184ab89ebc531a524ec009 | Impact of duration of surgery on pulmonary complications. After 10 h of surgery (cut off line) the probability of pulmonary complication significantly increased. | PMC10142708 | medicina-59-00722-g002.jpg |
0.436126 | 65bea74dc1664ff49da95e07893d7b90 | Cox cumulative analysis of mortality at 1 year. No statistical significance was noticed. | PMC10142708 | medicina-59-00722-g003.jpg |
0.555916 | 5d7c8362f63f451aac33dce9acc61af4 | Biosynthesis of major phytocannabinoids. OLS, olivetol synthase; OAC, olivetolic acid cyclase; GOT, geranylpyrophosphate:olivetolate geranyltransferase; CBDAS, cannabidiolic acid synthase; THCAS, tetrahydrocannabinolic acid synthase; CBDAS, cannabichromenic acid synthase; CBGA, cannabigerolic acid; CBDA, cannabidiolic acid; ∆9-THCA, ∆9-tetrahydrocannabinolic acid; CBCA, cannabichromenic acid; CBD, cannabidiol; ∆9-THC, ∆9-tetrahydrocannabinol; CBC, cannabichromene [11,12]. | PMC10142887 | plants-12-01664-g001.jpg |
0.495765 | 6e0fa219f3954a76a4541a99f55fb089 | Comparison of experimental variants of the cultivar ‘USO-31’ after 4 weeks of cultivation in Erlenmeyer flasks. It is visible that the control variant showed greater apical dominance and elongating shoot growth (A,C), while the growing variant on the medium with the presence of PEO-IAA showed a lower growth with limited apical dominance and more developed lateral shoots (B,D). | PMC10142887 | plants-12-01664-g002.jpg |
0.42985 | 10adc7beb62d400fa8246a01789eb803 | Comparison of experimental variants of the cultivar ‘Tatanka Pure CBD’ after 4 weeks of cultivation in Erlenmeyer flasks. Plant material at harvest before freezing with liquid nitrogen. The top shows the control variant (A). The bottom shows the experimental variant with the addition of PEO-IAA in the nutrient medium (B). The morphological differences between the variants are less visible in this cultivar than in the case of the ‘USO-31’ cultivar. | PMC10142887 | plants-12-01664-g003.jpg |
0.41002 | b935df6a247c45babb9ffff465176257 | Bar plots show mean values of the given RGEs (CBDAS, CBCAS, and OAC) of the experimental variant Cannabis sativa ‘USO 31’ (without the outliers) and ‘Tatanka Pure CBD’ cultivars. The points represent the concrete values. Expression data were normalised using Act as a housekeeping gene, and the experimental variant was calibrated relative to the control variant (RGEs of the control variants = 1). No statistically significant differences (at statistical significance level α = 0.05) were found between the analysed variants. | PMC10142887 | plants-12-01664-g004.jpg |
0.4922 | 516b9c50406f4b22ad5f3b238a31f11e | Bar plots show mean values and standard errors of the given concentration for both cultivars ‘USO-31’ and ‘Tatanka Pure CBD’. The points represent the concrete values. The matching samples are connected by lines. Significant differences in the concentrations of CBDA are represented by asterisks (p ≤ 0.05 “*”). | PMC10142887 | plants-12-01664-g005.jpg |
0.435217 | d434dfcd34464a018817078d06140f04 | The values of ln(RC) are plotted against the values ln(RGE). The number above the points indicates the number of the given sample. A positive correlation between ‘RC’ values and ‘RGE’ values can be observed. | PMC10142887 | plants-12-01664-g006.jpg |
0.386383 | b473e589e6ab4c34a78f16f647a1021f | Multiplication of the ‘USO-31’ cultivar on ViVi 6 culture medium to achieve the required amount of biomass to establish the experiment with PEO-IAA. | PMC10142887 | plants-12-01664-g007.jpg |
0.468629 | d20c697686f44c31a31e882662daa586 | Multiplication of the ‘Tatanka Pure CBD’ cultivar on ViVi 6 culture medium to achieve the required amount of biomass to establish the experiment with PEO-IAA. | PMC10142887 | plants-12-01664-g008.jpg |
0.419689 | d4cf11b998f94e20b42c52660e9274da | Flowchart illustrating the study’s design and the total number of individuals included. | PMC10143909 | viruses-15-00899-g001.jpg |
0.458446 | 8d5a8a383d9b456eb7b2cac4c03dc9c2 | Clinical characteristics of patients complaining of mild post-COVID-19 olfactory disorders. (A) absolute frequency of clinical symptoms observed during application of the clinical-epidemiological questionnaire (A, B, C, D, E, F, G) and otorhinolaryngological physical examination (H, I). The letters inside the circle represent the X-axis and indicate which group each variable belongs to. The lines between the groups represent the Y-axis and indicate the absolute frequency of each variable. (B) Absolute frequency of surgery and previous nasal trauma. * patients’ perception of symptoms. | PMC10143909 | viruses-15-00899-g002.jpg |
0.401827 | 58a92419ccc04c72b57f39b03167f2d1 | Nasal mucociliary mucosa of patients complaining of mild post-COVID-19 olfactory dysfunction. Negative control (A); positive control (B); patients (C–E). SARS-CoV-2 S/N proteins, in green; nucleic acid labeling (DAPI), in blue; ciliary structures-tubulin, in red. The “Merge” column shows all overlapping markups. Images (C–E) are representative of samples from 8 patients. The images were selected among a total of 24 images through three replicates. Scale bars represent 25 µm. Arrows indicate viral antigen (nucleocapsid and spike proteins) staining. | PMC10143909 | viruses-15-00899-g003.jpg |
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