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0.467387 | db8300e940c244938921d2ec7d55f318 | FCM detection of p-IκBα and IκBα in CMC cells cultured for 24 h by regular growth (control) or after treatment with DCF001 and/or TNFα under proliferative (PM) (blue peaks) or chondrogenic (CM) (red peaks) conditions. Data were reported as percentage expression (%) and p-IκB/IκBα expression ratio. The analysis was performed using indirect staining with primary antibodies against mouse anti-human p-IκBα or rabbit anti-human IκBα and Alexa Fluor® 488-conjugated secondary antibodies. Samples treated with only secondary antibodies were used as control (grey peaks). * p: vs. control; ^ p: vs. TNFα-primed cells. | PMC10299282 | ijms-24-10397-g005.jpg |
0.437104 | ba05711d16fd4cb897bd168c48cde0ed | Gene expression study of CMCs stimulated with DCF001 under proliferative (A) or chondrogenic (B) conditions. Cell cultures were challenged with TNFα (100 ng/mL) for 3 h before stimulation for a further 24 h with DCF001 solubilized in a PM or CM medium. Total RNA was extracted using the TRI Reagent solution and analyzed by quantitative real-time polymerase chain reaction (RT-qPCR). Data were reported as a relative fold increase in gene expression. * p: vs. control; ^ p: vs. TNFα-primed sample. | PMC10299282 | ijms-24-10397-g006.jpg |
0.414007 | 37e763219e7643fc9b6986390a3eafef | Summary of the study selection process for the systematic review and meta-analysis. | PMC10299436 | ijms-24-10136-g001.jpg |
0.469806 | 9629d5b071594e11a1f1cdaae807cf39 | Summary of quality assessment according to QUADAS-2 tool. Studies included in the systematic review are classified as at low-risk or high-risk of bias or applicability concerns for different domains (reported in the vertical axis). The horizontal axis indicates the percentage of studies. The graph indicates that a percentage above 60% was reached in all domains. | PMC10299436 | ijms-24-10136-g002.jpg |
0.415858 | c4f98f9d237842edb91e99a2702e585b | Meta-analysis and funnel plot concerning the detection rate of FAP-targeted PET in primary GC, [14,15,17,18,19,20,21,22]. | PMC10299436 | ijms-24-10136-g003.jpg |
0.433231 | 082a29faea24451784955469d9e2b07f | SROC curve of index test’s diagnostic accuracy in lymph node metastases. | PMC10299436 | ijms-24-10136-g004.jpg |
0.40955 | ecb69445198f4989a39236f1e9727a5e | Sensitivity and specificity of the index test in the assessment of lymph node metastases and relative forest plots. Legend: 95% C.I.: 95% confidence interval; TP: true positive; TN: true negative; FP: false positive; FN: false negative, [14,15,17,18,19,20,21,22]. | PMC10299436 | ijms-24-10136-g005.jpg |
0.445261 | 8625567b13fc48b8924e406be0fc6130 | Negative and positive likelihood ratios of the index test in the assessment of lymph node metastases and relative forest plots. Legend: 95% C.I.: 95% confidence interval; TP: true positive; TN: true negative; FP: false positive; FN: false negative, [14,15,17,18,19,20,21,22]. | PMC10299436 | ijms-24-10136-g006.jpg |
0.464397 | 921d5d0a5f104d29abb12957b5b0d3df | Diagnostic odds ratio of the index test in the assessment of lymph node metastases and relative forest plots. Legend: 95% C.I.: 95% confidence interval; TP: true positive; TN: true negative; FP: false positive; FN: false negative, [14,15,17,18,19,20,21,22]. | PMC10299436 | ijms-24-10136-g007.jpg |
0.452185 | 86cc161235284687aaf76cc480a820d5 | Meta-analysis and funnel plot concerning the detection rate of FAP-targeted PET in GC distant metastases, [14,15,17,18,19,20,21,22]. | PMC10299436 | ijms-24-10136-g008.jpg |
0.454391 | 567f1192ac244fe6a0730ec022323dbe | Pelvic TV ultrasound scan at 5 weeks of amenorrhea. The image shows an anechoic intrauterine scan compatible with a hemerion-free gestational chamber without cardiac activity and diffuse trophoblast characterized by an irregular profile. Both adnexa are normal. Reassessment is required after a week. | PMC10299875 | CRIOG2023-3752274.001.jpg |
0.394431 | 2f2fe8dbf34e4cd4b0579d60c2a7062e | Pelvic TV ultrasound at 6 weeks of amenorrhea in longitudinal (a), transverse (b), and magnified scan (c). Ultrasound scan show a small embrionary site without cardiac activity and a diffuse trophoblast with an irregular and breasted profile in its intracavitary portion. The pictures depose for internal abortion and suspected molar degeneration. | PMC10299875 | CRIOG2023-3752274.002.jpg |
0.440856 | b5f5e7e5d52b4c3ba016a7738f458752 | Pelvic TV ultrasound performed after the surgical procedure (D&C with suction evacuation), showing a regular thin endometrial line. | PMC10299875 | CRIOG2023-3752274.003.jpg |
0.470775 | 658a7a8dc2ff48aabff669cc9e33fc73 | Stormwater quality parameters during all rain events (Boxplots show the 2.5, 25, 50, 75, and 97.5 percentiles of all calculated EMCs); red lines represent the water quality objectives (WQOs), while green dot-dashed lines illustrate the maximum censored level (if any) for the OMPs. TOC and TSS in mg/L; conductivity (Cond) in µS/cm; and turbidity (Turb) in NTU | PMC10299930 | 11356_2023_27623_Fig1_HTML.jpg |
0.461437 | 2cb89da738854bfdb59cf5dc7a2117d8 | Non-exceedance probability (NEP) plots of the estimated EMCs for each OMP (black points are detects and red points censored data (non-detects); error bars show EMC errors (uncertainties); red lines represent the lowest PNEC levels for freshwater, based on water quality objectives (WQOs)) | PMC10299930 | 11356_2023_27623_Fig2_HTML.jpg |
0.459728 | fcab144685a04dc687c64837426f54b7 | Statistical analysis of EMC estimation errors (boxplots: min, Q25%, Q50%, Q75%, and max of relative EMC errors over N = 8 events) (orange bars: mean of relative analytical uncertainty observed for all events (EMAU or (δi/ci)mean)) | PMC10299930 | 11356_2023_27623_Fig3_HTML.jpg |
0.472238 | a5fb0cdfd863409292cec2a77e5bdf54 | Highly correlated parameters with OMPs in the studied catchment | PMC10299930 | 11356_2023_27623_Fig4_HTML.jpg |
0.396814 | 87cfba8558f6421e9301cc85b69a5036 | Composition-dependent phase evolution in Li2+xZrCl6-xOx.a XRD patterns of the mechanochemically synthesized Li2+xZrCl6-xOx. The broad hump below 30° comes from the Kapton film used to prevent air exposure during measurement. No smoothening was conducted to any of the diffraction data displayed here. b−d Variation of phase fractions (b), grain sizes (c), and crystallinity (d) with composition. The data were acquired from Rietveld refinement. | PMC10300059 | 41467_2023_39522_Fig1_HTML.jpg |
0.42199 | 644fdc7fe9be41c7ab4fff9998752876 | Li-ion transport behavior of Li2+xZrCl6-xOx.a Arrhenius plots of Li2+xZrCl6-xOx with different compositions. b Variation of the ionic conductivity (σ) at 25 °C and the activation energy (Ea) with x in Li2+xZrCl6-xOx. | PMC10300059 | 41467_2023_39522_Fig2_HTML.jpg |
0.423093 | 028323b2ccda452fad724f0c98811c35 | Structures and ionic conductivities of Li2.5-yZrCl5.5-yO0.5.a XRD patterns of Li2.5-yZrCl5.5-yO0.5 with different compositions. No smoothening was conducted to any of the diffraction data displayed here. b The Li2ZrCl6-Li4ZrCl4O2-LiZrCl5 ternary phase diagram with compositions A−D in (a) indicated by red dots. c Arrhenius plots of compositions A−D in (a). d Ionic conductivities at 25 °C and activation energies of compositions A−D in (a). | PMC10300059 | 41467_2023_39522_Fig3_HTML.jpg |
0.417075 | 0b43d1c836fe49acbb3208cb7923981b | Compressibility of LZCO.a−b SEM images with low (a) and high magnifications (b) for the cold-pressed pellets of Li10GeP2S12, Li6PS5Cl, Li2ZrCl6, Li3InCl6, and LZCO. All the pellets were fabricated under 300 MPa. The scale bars in (a and b) are 10 and 2 μm, respectively. c Comparison between the relative densities estimated by SEM and those estimated by comparing the actual density ρactual and theoretical density ρtheoretical. d Relative densities for the five solid electrolytes in (a and b), all of which were estimated from SEM. Each error bar was determined from the standard deviation of the data from five samples. | PMC10300059 | 41467_2023_39522_Fig4_HTML.jpg |
0.452273 | c115699975a54109b6628202a7bce7e0 | Electrochemical performance of the Li-In | LPSCl-LZCO | LCO cell.a Initial charge and discharge voltage profiles at 14 mA g–1, with the Coulombic efficiency ηCoulomb denoted. b, c Rate capability at 14, 28, 46.2, 70, and 140 mA g–1. d Long-term cycling performance at 700 mA g–1. All the cycling tests were conducted between 1.88 and 3.58 V vs. Li-In/Li+ at 25 °C and 1.5 tons of applied external pressure. | PMC10300059 | 41467_2023_39522_Fig5_HTML.jpg |
0.425046 | 82e20e1d4cc5455383574adbb09ba69b | Electrochemical performance of the Li-In | LPSCl-LZCO | scNMC811 cell.a Initial charge and discharge voltage profiles at 20 mA g–1, with the Coulombic efficiency ηCoulomb denoted. b, c Rate capability at 20, 40, 66, 100, and 200 mA g–1. d Long-term cycling performance at 1000 mA g–1. All the cycling tests were conducted between 2.18 and 3.68 V vs. Li-In/Li+ at 25 °C and 1.5 tons of applied external pressure. | PMC10300059 | 41467_2023_39522_Fig6_HTML.jpg |
0.467959 | a54f6512a72a46bc9943ef61ea1079a6 | a schematic representation of the main results.An adaptation of the stress-coping model by Maes et al.15. | PMC10300125 | 41531_2023_548_Fig1_HTML.jpg |
0.454737 | 255495ac896c418089095cb936e9c981 | (A) White and (B) brown sorghum types. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.) | PMC10300366 | gr1.jpg |
0.369057 | f870461662524aa7b613924eaee8c469 | Lesion location in each patient. The patient number (Pxx) was given according to the order of participation. Lesions were located mainly by diffusion-weighted imaging following ischemic stroke (P02, P04, P06–P15, P17–P19, P21, P23–P25, P27, and P28) or computed tomography (CT) scan following hemorrhagic stroke (P01, P05, P16, P20, P22, P26, P29, and P30). The lesion location in the ischemic stroke patient P03 was determined via CT scan because of a contraindication for using diffusion-weighted imaging. The left (Lt) side of the figure represents the right (Rt) side of the brain. | PMC10300420 | fnsys-17-1130272-g001.jpg |
0.432648 | c92fd4efed9241a3a89444188a88e992 | Experimental setup. (A) Functional near-infrared spectroscopy (fNIRS)-based neurofeedback training set up. The patient’s hand was hidden by a rack represented by a gray square on the illustration. The four red circles on the monitor indicated the visual stimuli that patients were required to remember. (B) Configuration of the fNIRS probe. Probes were placed over the prefrontal area. (C) Spatial registration of fNIRS maps onto Montreal Neurological Institute coordinates space. These feedback channels, ch.7 and ch.11, convey signals from the left and right dorsolateral prefrontal cortex (DLPFC) and frontopolar cortex (FPC), respectively. The profiles show typical ones of oxygenated hemoglobin (oxy-Hb), deoxygenated hemoglobin (deoxy-Hb), averaged z score and diameter of the blue circle, respectively. Time zero indicates the onset of the task block. During the task block, the oxy-Hb signals showed a greater response than the deoxy-Hb signals. As values for presenting neurofeedback information, the bilateral oxy-Hb signals were converted to z scores and the z scores were averaged in real-time. Finally, the diameter was calculated from the averaged z score. The size of the blue circle on the display increased only when the z score exceeded 1 and was not displayed during the Rest blocks. | PMC10300420 | fnsys-17-1130272-g002.jpg |
0.437993 | 6bd7756dcdef47c9ac82b864a46b2531 | Neuromodulation in the feedback channels (ch.7: upper panels, ch.11: lower panels) during the neurofeedback training task. (A) The temporal profiles of oxygenated hemoglobin (oxy-Hb) signals. Red and black lines represent the time courses of the oxy-Hb signals in the Real and Sham groups, respectively. The lighter-colored regions around the time course lines represent the standard deviation. The upper or lower directional standard deviation regions are shown for the profiles of the Real and Sham groups, respectively. In ch.11 of the Real group, clear task-related activity was observed in the final training session. (B) Beta value transitions. For ch.11 over the right prefrontal area, oxy-Hb beta values in the Real group increased gradually, whereas they decreased gradually in the Sham group. The beta value change of ch.11 in the Real group was consistent with the neurofeedback training aim, and the amount of change was significant even after Bonferroni correction. Error bars represent the standard deviation. (C) Between-group differences in the amount of beta value changes comparing first and sixth sessions. A significant difference was observed only in the right frontal area. †p < 0.1, *p < 0.05, **p < 0.01. | PMC10300420 | fnsys-17-1130272-g003.jpg |
0.467308 | 87ed648972a54e878884712b33cca22c | Behavioral performances reflecting working memory (WM) in the target-searching task. (A) Normalized movement distance transitions in the Pre- and Post-WM tasks. Red circle lines and black cross lines represent the Real and Sham groups, respectively. In both the Pre- and Post-WM task, patients successfully reduced the normalized movement distance, indicating optimization of the searching movement trajectory. (B) Mean normalized movement distance changes in the Real and Sham groups. Red circle lines and black cross lines represent the individual mean normalized movement distance values. Blue-filled circles indicate the patients with mild cognitive dysfunction in the Real group. The solid and dotted lines indicate improvement and worsening trends, respectively. The performance improved in 3/4 of the patients in the Real group, whereas improvement was observed in only one patient in the Sham group. Error bars represent standard deviations. The dagger and asterisks indicate the marginal and significant differences in the normalized movement distance between the Pre- and Post-WM tasks, respectively. †p < 0.1, **p < 0.01. | PMC10300420 | fnsys-17-1130272-g004.jpg |
0.413423 | 4a6b5806a45745f583f62f9f4e9a1b6a | Relationship between the individual beta value changes during neurofeedback training and the normalized movement distance changes from Pre-WM to Post-WM tasks. The lower-left panel shows the spatial configurations of the t-values from the correlation analyses. There was a significant negative correlation in ch.11 (right feedback channel), ch.10, and ch.15, but ch.7 (left feedback channel) did not show a significant correlation. The negative correlations indicate that patients achieving higher task-related activity during the neurofeedback training task also demonstrated higher behavioral performance improvement from the Pre-WM task to the Post-WM task. The blue-filled circles indicate the patients with cognitive dysfunction based on the Trail Making Test or the Behavioral Inattention Test. The asterisks indicate a significant correlation between the beta value changes and the normalized movement distance changes. In the lower-right panel showing the distribution of ch.11, the shape is changed according to the individual stroke lesion, and red and black shapes indicate the Real and Sham groups, respectively. *p < 0.05, **p < 0.01. | PMC10300420 | fnsys-17-1130272-g005.jpg |
0.393881 | 08543f22414a43d0aace521444fbd062 | Flowchart of the study design. A total of 60 patients and 120 teeth were evaluated in the in vitro and the in vivo clinical study. | PMC10300954 | medicina-59-01137-g001.jpg |
0.494537 | 3a92a5d659da45e1bfc28bb0eff5f4eb | Periapical X-ray of one sample from each group after shaping (left) and at the end of the root canal obturation (right): (a) Adseal; (b) Sealapex. | PMC10300954 | medicina-59-01137-g002.jpg |
0.456456 | b61f1480b36147b0929ff93a148ae069 | Images of two longitudinally sectioned samples from each group, showing the penetration of the methylene blue dye along the apical part of the root canal filling: (a) Adseal; (b) Sealapex. | PMC10300954 | medicina-59-01137-g003.jpg |
0.517518 | 2da1dcacbf7c4c348e1d2c7c5140b45b | Comparison of the degree of percolation measured in millimeters between patients treated with Adseal and Sealpex. | PMC10300954 | medicina-59-01137-g004.jpg |
0.491252 | 25638352efa140029cf5b93db9e6bb4b | Phenotypic test of pyrene biodegradation on a double-layer plate. Positive control: (1) M. vanbaalenii PYR-1 DSMZ 7251. Isolates: (2) MYC038, (3) MYC040, (4) MYC211, (5) MYC221, and (6) MYC223. | PMC10301208 | microorganisms-11-01413-g001.jpg |
0.543084 | 1d8656e21c25409a8b2d8883df233f01 | Pyrene biodegradation rates by MYC038, MYC040, MYC211, MYC221, and MYC223 isolates. (A) Pyrene biodegradation without peptone. (B) Pyrene biodegradation with 0.5% peptone. (C) CG/MS chromatograms showing biodegradation by MYC038 and MYC040 with peptone. T0h: black; T3d: blue; and T7d: red. | PMC10301208 | microorganisms-11-01413-g002a.jpg |
0.435497 | 10b3637acb7841538f66202b1650c983 | Schematic comparison of catabolic region A, responsible for PAH degradation, in M. vanbaalenii PYR-1 DSMZ 7251 and isolates MYC038, MYC040, MYC211, MYC221, and MYC223. In strain PYR-1, the start and end of region A are indicated by black arrows located at positions 494,420 and 642,917, respectively. This study analysed genes 1 to 5 (nidB2, nidB, nidA, nidA3, and nidB3). The ORFs found in the MYC isolates that matched the reference strain with 100% identity are colored red. ORFs with identity from 50% to 99% are marked in yellow or orange. ORFs with identity up to 50% are colored green, while ORFs that did not show identity are in gray. | PMC10301208 | microorganisms-11-01413-g003.jpg |
0.429259 | 7cd5f99081be4ae999b22219c0891c82 | Comparative genomic analysis based on protein sequence identity. MYC038 (1), MYC040 (2), MYC211 (3), MYC221 (4), MYC223 (5), M. sp JLS (6), M. sp MCS (7), M. tuberculosis H37Rv (8), and M. vanbaalenii PYR-1 (9). The blue-to-red color scale indicates the percentage identity between the proteins, ranging from 100% to 10%. Highlighted in red is the A region present in the isolates with reports of PAH degradation and absence of the negative control (M. tuberculosis H37Rv) and isolates in this study. | PMC10301208 | microorganisms-11-01413-g004.jpg |
0.446539 | acb7580574a3409cb87bd2c31da73c90 | Partial least−squares−discriminant analysis (PLS−DA) scores plot of the High HDL CAD (+) and High HDL CAD (−) groups. (A) Two−dimensional scores plot with 95% confidence regions. (B) Sparse three−dimensional scores plots. | PMC10301469 | metabolites-13-00695-g001.jpg |
0.40504 | 4736eb9cde14477483738f5b59483611 | (A) Volcano plot of the statistically significant lipids, derived from the comparison between CAD (+) and CAD (−) groups, generated by p value from Wilcoxon test and fold change (FC). Red plots indicate lipids are up−regulated in High HDL CAD (+) group and blue means down−regulated. Gray plots represent insignificantly altered lipids. (B) Heatmap visualization of significantly altered lipid species between the CAD (+) and CAD (−) groups. (C) Pathway analysis of differential lipids between the CAD (+) and CAD (−) groups. GPI= glycosylphosphatidylinositol; S1P = sphingosine−1−phosphate; PE = phosphatidylethanolamine; PS = phosphatidylserine; GlcCer = glucosylceramide; PG = phosphatidylglycerol; GM3 = monosialo−dihexosyl ganglioside. | PMC10301469 | metabolites-13-00695-g002.jpg |
0.453472 | 5c8d4af264d44c2f940cfb26fd6a8943 | Differential lipid correlation network using the Spearman correlation coefficients. PE = phosphatidylethanolamine; PS = phosphatidylserine; PG = phosphatidylglycerol; S1P = sphingosine−1−phosphate; GlcCer = glucosylceramide; GM3 = monosialo−dihexosyl ganglioside. | PMC10301469 | metabolites-13-00695-g003.jpg |
0.425449 | a263a2fb83eb4ea5aaeb0e36ae2e6145 | Selection and evaluation of lipid biomarker candidates of CAD with elevated HDL−C levels. (A) Screening potential lipid biomarkers using the AUC (x−axis) and value of specificity + sensitivity (y−axis) calculated by the ROC analysis. Three colored circles represent that AUC of differential lipids are greater than 0.8, and the sum of specificity and sensitivity are greater than 1.6 meanwhile. The white circles represent lipids with the sum of specificity and sensitivity less than 1.6. (B−D) ROC curves and histograms of GM3 (d18:1/22:0), GM3 (d18:0/22:0), and PS (38:4). (E) The ROC curve of the biomarker model generated by combining the three lipid biomarkers based on linear SVM. (F) The mean predicted class probabilities for individual samples across the one hundred cross−validations of the diagnostic model. GM3 = monosialo−dihexosyl ganglioside; PS = phosphatidylserine; AUC = area under the curve; Sens. = sensitivity; Spec. = specificity. | PMC10301469 | metabolites-13-00695-g004.jpg |
0.490055 | ab7499e1aa7c4fd79576599c1562149e | Schematic diagram of special anticancer drug administration route. | PMC10301590 | pharmaceutics-15-01664-g001.jpg |
0.43494 | 402bbc373efb4ab2934e161e7a6b84ea | Incidence of (A) black pod rot and (B) moniliasis (Moniliphthora roreri) in cacao pods under different treatments in the first bioassay. The treatments were regulation of shade and no regulation of shade. DCT, dicopper chloride trihydroxide. NMA1017, application of Paenibacillus sp. NMA1017. CONTROL, negative control with water. The asterisk indicates a statistical difference when compared with the negative control. | PMC10301746 | plants-12-02336-g001.jpg |
0.42268 | 3d69761b308247b89ac57d6895b1306f | Incidence of black pod rot in tagged cacao plants under different treatments in the second bioassay. DCT, dicopper chloride trihydroxide. NMA1017, application of Paenibacillus sp. NMA1017. CONTROL, negative control with water. The asterisks indicate statistical differences when compared with the negative control analyzed with the Tukey test. | PMC10301746 | plants-12-02336-g002.jpg |
0.445786 | b81394a93e864328a3ab551dfd62e1b2 | Incidence of moniliasis and black pod rot in tagged cacao trees under different treatments through time. (A) Incidence curve of moniliasis (Moniliophthora roreri). (B) Incidence curve of black pod rot (Phytophthora tropicalis). DCT, dicopper chloride trihydroxide. NMA1017, application of Paenibacillus sp. NMA1017. CONTROL, negative control with water. The evaluation dates were every 15 days. | PMC10301746 | plants-12-02336-g003.jpg |
0.425104 | bac1ed0f2137402a9d9ddd4580265716 | Incidence of moniliasis (Moniliophthora roreri) in tagged cacao pods under different treatments in the second bioassay. The treatments were: DCT, dicopper chloride trihydroxide; NMA1017, application of Paenibacillus sp. NMA1017; and CONTROL, negative control with water. | PMC10301746 | plants-12-02336-g004.jpg |
0.464169 | 9c0cb3de1e9344f7b61d34f11073ad53 | Graphical incidence of moniliasis (Moniliophthora roreri) in tagged cacao pods under different treatments in the second bioassay. DCT, dicopper chloride trihydroxide. NMA1017, application of Paenibacillus sp. NMA1017. CONTROL, negative control with water. The asterisk indicates statistical differences when compared with the negative control. | PMC10301746 | plants-12-02336-g005.jpg |
0.402331 | e16a07a753254d479d3f90785c157cf7 | External (A–C) and internal (D–F) severities of moniliasis (Moniliophthora roreri) in tagged cacao pods. The treatments were: DCT, dicopper chloride trihydroxide; NMA1017, application of Paenibacillus sp. NMA1017; and CONTROL, negative control with water. | PMC10301746 | plants-12-02336-g006.jpg |
0.441672 | acf9bb1e591c4bfc8ba7c7ba01f401e5 | Graphic of external (A) and internal (B) severities of moniliasis (Moniliophthora roreri) in tagged cacao pods. The treatments were: DCT, dicopper chloride trihydroxide; NMA1017, application of Paenibacillus sp. NMA1017; and CONTROL, negative control with water. The asterisk indicates statistical differences when compared with the negative control. | PMC10301746 | plants-12-02336-g007.jpg |
0.41525 | 48720c917695408a8ae74357397730fe | Cacao tree (A) with a trapping device (B) and Moniliophthora roreri spores (C) observed at 10×. | PMC10301746 | plants-12-02336-g008.jpg |
0.426651 | bca8787b8e23458499254e9fa6c1c706 | Visual representation of polyHEMA network prepared with water and DES and their sol-gel transitions. | PMC10302097 | polymers-15-02605-g001.jpg |
0.514873 | c566757ba7b1425fa3c4f2707a1a6028 | UV-vis spectrum of hydrogels (A) and DES gels (B) with different weight ratios of HEMA. | PMC10302097 | polymers-15-02605-g002.jpg |
0.542633 | 3cd587e95cd0478d8c2fa1c6cf87262e | ATR-FTIR spectra (A) and enlarged wavenumber at 2000–600 cm−1 of a polyHEMA hydrogel (a to c), a DES gel (d to f), and a HEMA/PEGDA co-monomer mixture (g). The vertical dashed line indicates the position of the peaks associated with the polymerizable methacrylate/acrylate group. (B) The peaks disappeared as the reaction time increased, indicating complete copolymerization. Polymerization time: 1.5 h (c & f); 6 h (b & e); 24 h (a & d). | PMC10302097 | polymers-15-02605-g003.jpg |
0.497102 | 26f99b0cab9c44dfb445f265e88a62a4 | Images of fragile hydrogels (A) and tough DES gels (B) before and after 75% compression. | PMC10302097 | polymers-15-02605-g004a.jpg |
0.464784 | c31530975c604d2280e87df5baba6763 | Compressive stress−strain curves of polyHEMA hydrogels (A) and DES gels (B) with different weight ratios of HEMA. (C) Young’s moduli of the DES gels with different weight ratios of HEMA. (D) Compressive stress−strain curves of D5 gel with varying maximum compression under loading–unloading cycles. (E) Compressive stress–strain curves of D5 gel at 90% strain for three loading–unloading cycles. | PMC10302097 | polymers-15-02605-g005a.jpg |
0.509894 | 77c8a6137cdb46d5b5fbc97c1ad757bd | (A) Tensile stress−strain curves of DES gels with different weight ratios of HEMA. (B) Young’s moduli of DES gels with different weight ratios of HEMA. (C) Relationship of the stress and strain at break values with the different weight ratios of HEMA in DES gels. | PMC10302097 | polymers-15-02605-g006.jpg |
0.39828 | 8166e96ae8d243eda8b53b9fbbe250e4 | (A) Photographs of contact lenses prepared using the W5 hydrogel and the D5 gel before and after 24 h in air. (B) Remaining weight of W5 hydrogel and D5 gel was in a constant humidity (64%) and constant temperature (25 °C) environment for 24 h. | PMC10302097 | polymers-15-02605-g007a.jpg |
0.441175 | 3ac73fbbe0f64fea9c6ba2680c095f35 | LED emission testing in an electrical circuit connected in series with W5 hydrogel and D5 gel. | PMC10302097 | polymers-15-02605-g008.jpg |
0.428315 | 1e577a04c1cc491c9d761c4b64169e38 | Adsorption of PDA on MNPs. (A) Representative images of dopamine after polymerization reaction (PDA) and MNPs before and after coupling (PDA–MNPs). (B) Values of PDA (polymerized in situ) measured at 550 nm. The percentage ratio between PDA and PDA–MNPs or MNPs alone is shown. (C) Particle size distribution of the prepared PDA-MNPs along with the bare MNPs and PDA, measured by dynamic light scattering (DLS). (D) ζ-potential of MNPs, PDA, and PDA–MNPs detected at pH 7.4. The results were obtained from three independent experiments made in triplicate. Differences between groups were assessed by one-way ANOVA with Bonferroni’s multiple comparison post-test (*** p ≤ 0.001). | PMC10302170 | pharmaceutics-15-01711-g001.jpg |
0.414093 | 958be60563fb4ca188f3d33c6aa9bacd | Characterization of primary macrophages from mouse peritoneal exudate (PEMs) by flow cytometry analysis. (A) Representative flow cytometry dot plot of the physical parameters of PEMs. FSC: forward scatter (size); SSC: side scatter (granularity). (B–D) The population of PEMs was characterized using a combination of fluorochrome-conjugated antibodies typical of several immune cell populations: CD45+ F4/80+ (macrophages, Mϕ), CD11b (myeloid cells), Gr1 (granulocytes), CD19 (B cells), and CD3 (T cells). PEMs were F4/80+ CD11b+, and negative for Gr1, CD19, and CD3. (E,F) The CD45+ F4/80− population included monocytes and B cells. The results are representative of three independent experiments. | PMC10302170 | pharmaceutics-15-01711-g002.jpg |
0.411514 | fdb7e1e4a7f34274bd26bb2ed509fe6a | Cytocompatibility of PDA–MNPs on PEM cells. (A) Cell viability was assessed by MTT assays after cells were incubated with PDA–MNPs at different concentrations for 72 h. Untreated cells were taken as a reference value (100%), while exposure to H2O2 at 1 µM represented the positive control. Data are expressed as the mean ± SD of at least four independent experiments performed in triplicate. (B) Immunofluorescence images showing reactive oxygen species (ROS) production in PEM cells incubated with PDA–MNPs (100 μg/mL) for different time points (from 4 to 24 h). Fixed and permeabilized cells were stained with TRITC-phalloidin (red) for actin and DAPI (blue) for nuclei, while ROS production was visualized in green. LPS/INF-γ 24 h treated cells were used as a positive control. Magnification, 200×. (C) Histograms showing the expression of ER stress markers (Bip, XBP1s, ATF4, and ATF6) assessed by q–RT–PCR on PEM cells incubated with PDA–MNPs (100 μg/mL) in the same conditions as above. ER stress was mostly observed in cells treated with 2 μg/mL tunicamycin for 8 h, used as a positive control. Gene expression is shown as a fold change relative to untreated cells. The data are expressed as the mean ± SD of three independent experiments. Statistical analyses were carried out using one-way ANOVA with the Bonferroni comparison post-test vs. the untreated controls (* p ≤ 0.05; ** p ≤ 0.01; *** p ≤ 0.001; **** p < 0.0001). | PMC10302170 | pharmaceutics-15-01711-g003.jpg |
0.407462 | 7ff09434364745da8de49dca3dc6c438 | Interaction of PDA–MNPs with PEMs in the presence/absence of a gradient magnetic field (GMF). (A) Images show PEMs treated with PDA–MNPs and stained Prussian blue and nuclear fast red. Magnification 200×. (B) Amount of iron associated with cells as quantified with potassium thiocyanate: cells were incubated with PDA–MNPs for different time points (from 0.5 to 30 min) in the absence (−GMF) or in the presence (+GMF) of a gradient magnetic field. Untreated cells were used as a negative control. The results are expressed as mean µg/mL ± SD and were obtained from at least three independent experiments performed in triplicate. (C) Representative images showing the granularity and size of untreated control cells (left) or cells treated with 20 μg of PDA–MNPs for 24 h. Merge panels are shown on the right and were used for calculations. The data were analyzed by a t-test for each time point (*** p < 0.001). | PMC10302170 | pharmaceutics-15-01711-g004.jpg |
0.425595 | 20c5e74e3c634e21bb7259a9fd3ef559 | Preparation and the cellular uptake of PDA–MNPs/siRNA in PEM cells. (A) Images showing the loading of siRNA onto nanocarriers examined by agarose gel electrophoresis. Different ratios of MNPs and PDA–MNPs with 10 µM siRNA were used in this typical experiment. (B) Quantification of the percentage of siRNA bound to MNPs or PDA–MNPs, obtained by subtracting the amount of unbound siRNA from the total siRNA after magnetic decantation (densitometric analysis). The results are expressed as % absorption ± SD of the percentage (delta method); they were compared among them for statistical analysis and were obtained from three independent experiments performed in triplicate. (C) Delivery of the siRNAs loaded on nanoparticles into PEMs in the presence/absence of GMF obtained by fluorescence microscopy. Cells were incubated with FITC–labeled siRNA complexes (20 µg) for 24 h. Images were acquired after fixation, permeabilization, and staining for nuclei with DAPI (blue). siRNA transfection with Lipofectamine 2000 (LIPO) and PDA–MNPs alone represent the controls. (D) Cell uptake efficiency in PEMs, measured by flow cytometry. The values of FITC–siRNAs are indicated for each treatment. Statistical analyses were performed using a t–test for each experimental concentration (*** p < 0.001). | PMC10302170 | pharmaceutics-15-01711-g005.jpg |
0.438174 | d4c4603b3bbe41b8aadd059642d1f191 | Gene silencing by siRNA delivered by nanoparticles in PEMs. (A) WB analysis of the silencing of PERK and its downstream effector phospho–eiF2–α in PEMs treated with LIPO/siPERK and PDAMNPs/siPERK for 48 h. The ER stress inducer tunicamycin (TM, 2 µg/mL) was used as a control along with PDA–MNPs alone. (B) Densitometric analysis of protein bands using the protein expression of α-Tubulin as an internal control. (C,D) Histograms showing mRNA levels of phospho–eiF2–α downstream effectors (ATF4 and CHOP) and ER stress sensors (ATF6 and XBP1) detected by q–RT–PCR following the silencing of PERK in PEM cells with PDA-MNPs/siPERK using LIPO/siPERK as a positive control for gene silencing. Gene expression was expressed as a fold change relative to untreated cells. The data are expressed as the mean ± SD of three independent experiments using one-way ANOVA with the Bonferroni comparison post-test vs. the untreated controls (* p ≤ 0.05; ** p ≤ 0.01; *** p ≤ 0.001; **** p < 0.0001). | PMC10302170 | pharmaceutics-15-01711-g006.jpg |
0.409307 | fd2a164fab754bd7a652ba8af7711758 | PEMs can be polarized in vitro towards the M1 or M2 phenotypes. (A) Images of PEMs after isolation and culture for 5 days with the macrophage colony-stimulating factor (M–CSF) and stimulation for a further 48 h, either with LPS/IFN–γ or IL-4 to induce the M1 and M2 phenotypes, respectively. (B) Relative expression of specific genes for M1 and M2 phenotypes was determined by q–RT–PCR. Gene expression was expressed as a fold change relative to untreated M0 cells. The data are expressed as the fold-change expression ± SD of three independent experiments. (C) Immunofluorescence images of differentiated PEMs into M1 and M2 phenotypes. Cells were fixed, permeabilized, and stained with FITC-streptavidin F4/80 antibody for macrophage identification (red) and DAPI (blue) for the nuclei. The expression of CD86 (upper lane) and CD206 (lower lane) was visualized by green fluorescence. Images were taken at 200x magnification. (D) FACS analyses of the expression of F4/80 (FITC), CD86 (PE), and CD206 (PE/Cy7), markers of macrophages in M0, M1, and M2, respectively. Statistical analyses were performed using one-way ANOVA with Bonferroni’s multiple comparison post-test (* p ≤ 0.05; ** p ≤ 0.01; **** p < 0.0001). | PMC10302170 | pharmaceutics-15-01711-g007.jpg |
0.410861 | cf5409d5b8cc4d1a97e2b841ce1ed0cb | Reprogramming of TAMs from the M2 to the M1 phenotype. PEMs were polarized to the M2 phenotype with IL-4, treated with ER stress sensor tunicamycin (2 µg/mL), and incubated with LIPO/siPERK and PDA–MNPs/siPERK. (A) After 24 h of incubation, the mRNA levels of the genes specific for M1 or M2 phenotypes were determined by q–RT–PCR. (B) WB representing the expression and state of activation in M2/TM (TAM–like) of NF-κB p65, STAT3, and MAPK1/2, 48 h after silencing PERK mRNA. Vinculin was used as the loading control. (C) Images showing the expression of CD86 (top row) and CD206 (bottom row) observed using fluorescence microscopy after M2/TM were incubated for 3 days with LIPO/siPERK and PDA–MNPs/siPERK, fixed, permeabilized, and stained with FITC–labeled antibodies and with DAPI (blue) for nuclei. Magnification: 200×. (D) Histograms showing the quantification of CD86 and CD206 expression determined by flow cytometry analysis after M2/TM were incubated for 3 days with LIPO/siPERK and PDA-MNPs/siPERK. The results for q–RT–PCR are expressed as the fold change ± SD with respect to the untreated M2/TM, obtained in three independent experiments made in triplicate and analyzed by one-way ANOVA with Bonferroni’s multiple comparison test (* p ≤ 0.05; ** p ≤ 0.01; **** p < 0.0001). | PMC10302170 | pharmaceutics-15-01711-g008.jpg |
0.351054 | db1bebb7d71743a18f48cd50f1d5f881 | Effect of terminal drought stress on (A) RWC and (B) CTD of studied chickpea genotypes, where RWC and CTD indicate relative water content and canopy temperature depression, respectively. | PMC10302310 | life-13-01405-g001.jpg |
0.446732 | e32fc360c5314014bc8f0cdbb4e1d47c | Various topics that could be discussed in a review of the “Common cold”. | PMC10324571 | falgy-04-1224988-g001.jpg |
0.509505 | c47f2cc213fb4cb4a3283d3bd6f39d60 | Iceberg concept of viral infection. Most infections result in asymptomatic or mild symptoms which may not reach a threshold to be self-diagnosed as a common cold. These infections are hidden and hence “below the water line of an iceberg”. Severe symptoms may reach a threshold to be termed “flu- like illness”and lower respiratory tract infections such as bronchiolitis and pneumonia may not be appreciated as often starting as a common cold. | PMC10324571 | falgy-04-1224988-g002.jpg |
0.424646 | 287313346b504221b36dc0ff13d090e4 | Factors that may influence susceptibility and incidence of common cold. | PMC10324571 | falgy-04-1224988-g003.jpg |
0.442572 | 5d0ef40050304068aeb821a63615f331 | Mechanism of symptoms. | PMC10324571 | falgy-04-1224988-g004.jpg |
0.446479 | d3d71d82d4be4f7a95ee270b323d5c0d | The procedure for selecting the studies included in this meta-analysis. RA: rheumatoid arthritis. | PMC10324936 | jrd-30-2-116-f1.jpg |
0.426816 | c4ca7d9b340e4838bd2a448c5869f3b2 | Meta-analysis investigating the relationship between leptin and RA, taking into account all study participants (A) and considering any racial groups (B). CI: confidence interval, RA: rheumatoid arthritis. | PMC10324936 | jrd-30-2-116-f2.jpg |
0.407076 | d05f73296074453689a1dadc20c80e26 | Meta-analysis of the relationship between leptin and the DAS28-ESR (A) as well as CRP (B). CI: confidence interval, DAS28-ESR: Disease Activity Score 28-erythrocyte sedimentation rate, CRP: C-reactive protein. | PMC10324936 | jrd-30-2-116-f3.jpg |
0.54741 | 2dd32cde70c34889b1f1e5adbe9937c2 | The funnel plot displaying the results of the studies that investigated the connection between RA and leptin levels. The filled circles indicate the studies that demonstrate publication bias. The diamonds at the bottom of the graph represent the summary estimates of the impact, both before and after accounting for publication bias. RA: rheumatoid arthritis. | PMC10324936 | jrd-30-2-116-f4.jpg |
0.471292 | c5e7fad8e5164b19bcd6a1cdf0627928 | Illustration of a Transcatheter Single Chamber Pacing System Positioned in the Right Ventricular Apex | PMC10326662 | aer-12-e09-g001.jpg |
0.425501 | 83500a95f4524d72acc82870f509ef7a | Micra Electrical Parameters by Study Visit | PMC10326662 | aer-12-e09-g002.jpg |
0.459342 | 4c07fc84eba04db48cfda7e9e31c2547 | Medtronic Micra-AV Accelerometer Signals | PMC10326662 | aer-12-e09-g003.jpg |
0.409042 | f24587c01b8d47dca2a07d0ad88f37cd | Components of the WiSE CRT System: Battery and Ultrasound Transmitter Implanted Subcutaneously | PMC10326662 | aer-12-e09-g004.jpg |
0.472807 | 2072d48f2a0e4d01be1c05221e4ea325 | Micra Extraction | PMC10326662 | aer-12-e09-g005.jpg |
0.448665 | c0cae907bb5740b699d8a5ca79cd8d85 | T. cruzi infection impacts the urine metabolome more than the salivary and plasma metabolome.(A) Parasite burden at heart base decreases as mice transition from acute to chronic infection. (B) Urine sample principal coordinate analysis. (C) Urine sample volatility analysis showing separation between infected samples and both uninfected and isoproterenol-treated samples. Thick lines indicate group mean and thin lines represent the trajectory of each individual mouse along principal coordinate axis 2. *, PERMANOVA p<0.05 for infected to uninfected animals. (D) Saliva sample principal coordinate analysis. (E) Plasma sample principal coordinate analysis. | PMC10326868 | nihpp-2023.06.03.543565v1-f0002.jpg |
0.42701 | 2ab51febcda24f5e8216310f65d614b6 | Lack of commonalities between biofluids or in a given biofluid in response to isoproterenol treatment or T. cruzi infection, based on GLMM output.(A) Limited overlap of metabolites perturbed by T. cruzi infection between biofluids. (B) No overlap of metabolites perturbed by isoproterenol treatment between biofluids. (C) No commonalities between metabolites significantly perturbed by infection or isoproterenol treatment in plasma samples. (D) Limited commonality between significantly perturbed metabolites in saliva samples. (E) No commonalities between significantly perturbed metabolites in urine samples. | PMC10326868 | nihpp-2023.06.03.543565v1-f0003.jpg |
0.426168 | 35a8f46ceb764eae835dbac361232b2b | Strong commonalities between biofluids in terms of metabolite occurrence.(A) Large overlap of metabolites present in infected biofluids. (B) Large overlap of metabolites present following isoproterenol injection in biofluids. (C) Large commonalities found between metabolites in infected or isoproterenol treated plasma samples. (D) Large commonalities between metabolites in infected or isoproterenol treated saliva samples. (E) Large commonalities between metabolites in infected or isoproterenol treated urine samples. | PMC10326868 | nihpp-2023.06.03.543565v1-f0004.jpg |
0.551633 | f9f32c853e3d4156b29fb73a11b10303 | Representative ROC curves (validation cohort). | PMC10326868 | nihpp-2023.06.03.543565v1-f0005.jpg |
0.485355 | f018a52a12ac4de29d5fbd38509b4b82 | Urine metabolome in mice that successfully cleared T. cruzi shows no significant difference from mice in which parasites failed to be cleared.(A) Cardiac parasite burden post-infection, treatment and immunosuppression in mice in which parasites persisted (failures) versus those where parasites were successfully cleared (success), as defined by our cutoff compared to qPCR background in uninfected samples. (B) Urine sample principal coordinate analysis for mice that were infected and then treated (irrespective of successful or failed parasite clearance) versus uninfected control mice. Samples collected prior to immunosuppression. PERMANOVA p < 0.05 for treated mice versus uninfected mice, R2 = 0.08. (C) Same PCoA analysis as in (B), recolored to compare successfully-treated mice, mice where treatment failed to clear parasites, and uninfected control mice. PERMANOVA p > 0.05 for successful parasite clearance versus failed parasite clearance, R2 = 0.03. (D) Same PCoA analysis as in (B), recolored to display normalized cardiac parasite burden post-immunosuppression, R2 = 0.02. No segregation by parasite burden was observed. Uninfected controls enlarged for contrast. | PMC10326868 | nihpp-2023.06.03.543565v1-f0006.jpg |
0.423264 | d6b44625c5fb41fbb33a5b7810375a46 | Metabolite features previously identified as differing between infected and uninfected samples remain significantly perturbed even after successful parasite clearance.Significance was established using p-values determined by a Kruskal-Wallis test. P-values were corrected using a Dunn Test with Benjamini-Hochberg adjustment to control the false discovery rate; FDR-corrected p < 0.05 = *, FDR-corrected p < 0.01 = **, FDR-corrected p < 0.001 = ***. | PMC10326868 | nihpp-2023.06.03.543565v1-f0007.jpg |
0.417042 | e2e164274a734390a836dc3107e5927d | Synthetic NET (sNET) formulation.(A) Schematic of sNET formation by combining 50 wt% DNA and 50 wt% histones dissolved separately in buffer. The DNA and histone will electrostatically interact to form large complexes that can be sonicated to create a suspension of sNETs. (B) Fluorescence microscopy image of the DNA structure of an sNET, stained with DAPI. Scale bar, 25 µm. | PMC10327088 | nihpp-2023.06.26.546583v1-f0001.jpg |
0.436343 | 50385101c1ca414280173fac45b6541a | sNETs Alter Synthetic Mucus Microstructure.(A) Schematic of synthetic mucus hydrogel preparation with various additives (sNETs, DNA, Histones, or additional PGM). (B) Representative trajectories of PEGylated 100 nm nanoparticle diffusing within each hydrogel type for 1.5 seconds. Scale bar, 200 nm. Box and whisker plots of (C) the logarithm based 10 of the measured MSD at 1 second (log10[MSD1s]) of PEGylated 100 nm nanoparticles in each hydrogel type (n = 3 replicates per condition, with 5 different randomly chosen regions of each individual gel imaged), and (D) the estimated pore size of each hydrogel type calculated based on measured MSD. (E) Box and whisker plot of the log10[MSD1s] of 100 nm PEGylated nanoparticles measured for each hydrogel type with and without 7 µg/ml DNase I treatment for 1 hour at 37°C (n = 3 replicates per condition, with 5 different randomly chosen regions of each individual gel imaged). For (C–E), statistical significance determined by Kruskall Wallis test with Dunn’s multiple comparisons test (**** = p < 0.0001). | PMC10327088 | nihpp-2023.06.26.546583v1-f0002.jpg |
0.435002 | ae7d896e98e045c5a53982320310f648 | sNETs Increase Synthetic Mucus Viscoelasticity.(A) Mean elastic, (B) viscous moduli (G′, G″) and (C) complex viscosity (η*) at ω = 1 rad/s for SM and sNET-SM hydrogels (n = 3 replicates per condition). Statistical significance determined by unpaired two tailed t-test (** = p < 0.01). | PMC10327088 | nihpp-2023.06.26.546583v1-f0003.jpg |
0.421435 | 8c1b8a965c9e4410bce012284cc38442 | sNETs Decrease Mucociliary Transport In Vitro.(A) Schematic of the process of washing cultures to harvest HAE mucus, then incorporating sNETs into the fresh HAE mucus (sNET+ HAE mucus), and overlaying the sNET+ HAE mucus back onto the surface of the cultures to measure MCT using fluorescent microspheres. (B) Median transport rates of the 2 µm microspheres and (C) the mean cilia beat frequency of cultures overlaid with control and sNET+ HAE mucus (n = 3 replicates per condition, with 3 randomly chosen regions of each individual culture imaged). (D) Representative trajectories of the microspheres being transported across the mucosal surface of cultures over the course of 10 seconds with control or sNET+ HAE mucus applied. Scale bar, 100 µm. (E) Median transport rates and (F) mean cilia beat frequency measured from the same set of cultures with either control or sNET+ HAE mucus overlaid before (pretreatment) and after (DNase treated) application of DNase I at a concentration of 7 µg/ml for 30 minutes at 37°C (n = 3 replicates per condition, with 3 randomly chosen regions of each individual culture imaged). Statistical significance determined by unpaired two tailed t-test for (B, C), a two-way repeated measures ANOVA test for (E, F) (Ns = p > 0.05, * = p < 0.05). | PMC10327088 | nihpp-2023.06.26.546583v1-f0004.jpg |
0.458822 | edc280d58e67438a9a89ba9078585040 | sNETs Alter Bacterial Growth Patterns and Sustain Survival in Mucus.Fluorescence microscopy images of GFP-expressing PAO1 (GFP PAO1) bacteria grown for 24 hours at 37°C in (A) control or (B) sNET+ HAE mucus. Scale bar, 100 µm. GFP PAO1 were grown in (C) control or (D) sNET+ HAE mucus for 24 hours and the GFP fluorescence of the bacteria was measured at 0, 3, 6, and 24 hours (n = 3 replicates per condition). Values are plotted as percentage of the baseline fluorescence at 0 h with background fluorescence subtracted from wells containing either control or sNET+ HAE mucus with no bacteria. Statistical significance determined by one-way repeated measures ANOVA tests with Tukey’s multiple comparisons tests (Ns = p > 0.05, * = p < 0.05, **** = p < 0.0001). | PMC10327088 | nihpp-2023.06.26.546583v1-f0005.jpg |
0.421096 | 21c166d1b4c844879c27b6a366e6f082 | Flowchart of included cases. | PMC10327580 | ActaO-94-13650-g001.jpg |
0.401907 | b3abef9fc34b424a89794c42fb8ad53c | (A, left) Incidence per 105 person-years for Achilles, quadriceps, and hamstring injuries (ICD-10 S86.0, S76.1, and S76.3) and (B, right) incidence of operative treatment of the respective injuries for all Swedish adults aged between 18 and 90 years between 2001 and 2020. | PMC10327580 | ActaO-94-13650-g002.jpg |
0.41328 | 6bed26666bd24d0f8eb39dd709823d86 | (A, left) Age distribution and (B, right) seasonal variation for time of injury for male and female patients with hamstring injuries (ICD-10 S76.3) in Sweden between 2001 and 2020. | PMC10327580 | ActaO-94-13650-g003.jpg |
0.420111 | eef8dca931914ea1af47db16d7288cf6 | Incidence of hamstring injuries (ICD-10 S76.3) in Swedish adults per 105 person-years between 2001 and 2020 for (A, left) men and (B, right) women by age group. | PMC10327580 | ActaO-94-13650-g004.jpg |
0.39505 | 3a1a0f9c848a4f33b9edeb58d71f1f13 | Proportion of patients with (A, left) hamstring injuries (ICD-10 S76.3) operated on in Sweden between 2001 and 2020 by age group, and (B, right) hamstring injuries (ICD-10 S76.3) operated on with the primary contact in high- or low- volume centers between 2001 and 2020. | PMC10327580 | ActaO-94-13650-g005.jpg |
0.399781 | c3196871ede648e2af016d04622afcc4 | Example images from the datasets used in this study: (a) synthetic dataset example with added ring, (b) synthetic dataset without ring, (c) BBBC010 dataset example with mostly alive C. elegans, (d) BBBC010 dataset patch with mostly dead C. elegans, (e) mating dataset with petri-dish ring, (f) zoomed-in mating dataset patch with many overlaps. | PMC10328995 | 41598_2023_38213_Fig1_HTML.jpg |
0.443314 | 80c5d81748f843c88abd8d5bf1641d31 | Network architecture based on Swin-L backbone and HTC. Batch norm (BN) layers in HTC are replaced by group norm (GN) + weight standardization (WS). Bounding box heads are changed from the original Shared2FC architecture to Shared4Conv1FC. | PMC10328995 | 41598_2023_38213_Fig2_HTML.jpg |
0.463485 | 88ac86e50a804a8cb131c77d0adb62fb | Example from the CSB-1 dataset (box and mask colors are selected randomly). (a) Ground truth annotations, (b) predicted bounding boxes and masks, (c) TP (green), FP and FN (red) pixels. | PMC10328995 | 41598_2023_38213_Fig3_HTML.jpg |
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