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Is mannose-binding lectin deficiency inherited ?
The inheritance pattern of mannose-binding lectin deficiency is unclear. Some reports show that having a disease-associated mutation in one copy of the MBL2 gene in each cell can lead to the condition, while other reports state that a mutation in both copies of the gene is necessary. It is important to note that people inherit an increased risk of developing mannose-binding lectin deficiency, not the condition itself. Not all people who inherit mutations in this gene will develop the condition.
96,955
What are the treatments for Hereditary sensory neuropathy type 1 ?
How might hereditary sensory neuropathy type 1 be treated? Management of hereditary sensory neuropathy type 1 generally follows the guidelines for diabetic foot care, including careful cleansing and protection of wounds and surgical care when needed. Pain medications may be used by those who experience shooting pains.
101,671
What area of study did professor Mark Long have a degree in?
economics
55,128
What are the treatments for autosomal recessive axonal neuropathy with neuromyotonia ?
These resources address the diagnosis or management of autosomal recessive axonal neuropathy with neuromyotonia: - Genetic Testing Registry: Gamstorp-Wohlfart syndrome These resources from MedlinePlus offer information about the diagnosis and management of various health conditions: - Diagnostic Tests - Drug Therapy - Surgery and Rehabilitation - Genetic Counseling - Palliative Care
97,271
How many miles of trails are in The Loop?
over 100
76,857
Is polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy inherited ?
This condition is inherited in an autosomal recessive pattern, which means both copies of the gene in each cell have mutations. The parents of an individual with an autosomal recessive condition each carry one copy of the mutated gene, but they typically do not show signs and symptoms of the condition.
97,410
What group was established in response to Executive Order 9808?
President's Committee on Civil Rights
54,950
How many households were the offices of Wanhu in charge of?
10,000 households
2,268
What does the acronym LES refer to?
source luminous efficacy
40,872
What did the second Famicom controller have as a unique feature?
microphone
35,390
During what king's reign did 60 Protestants die for heresy?
Henry VIII
11,930
What states formed Berlin in 1990?
West and East Berlin
41,362
What are the genetic changes related to Diamond-Blackfan anemia ?
Diamond-Blackfan anemia can be caused by mutations in the RPL5, RPL11, RPL35A, RPS7, RPS10, RPS17, RPS19, RPS24, and RPS26 genes. These genes provide instructions for making several of the approximately 80 different ribosomal proteins, which are components of cellular structures called ribosomes. Ribosomes process the cell's genetic instructions to create proteins. Each ribosome is made up of two parts (subunits) called the large and small subunits. The RPL5, RPL11, and RPL35A genes provide instructions for making ribosomal proteins that are among those found in the large subunit. The ribosomal proteins produced from the RPS7, RPS10, RPS17, RPS19, RPS24, and RPS26 genes are among those found in the small subunit. The specific functions of each ribosomal protein within these subunits are unclear. Some ribosomal proteins are involved in the assembly or stability of ribosomes. Others help carry out the ribosome's main function of building new proteins. Studies suggest that some ribosomal proteins may have other functions, such as participating in chemical signaling pathways within the cell, regulating cell division, and controlling the self-destruction of cells (apoptosis). Mutations in any of the genes listed above are believed to affect the stability or function of the ribosomal proteins. Studies indicate that a shortage of functioning ribosomal proteins may increase the self-destruction of blood-forming cells in the bone marrow, resulting in anemia. Abnormal regulation of cell division or inappropriate triggering of apoptosis may contribute to the other health problems that affect some people with Diamond-Blackfan anemia. Approximately 25 percent of individuals with Diamond-Blackfan anemia have identified mutations in the RPS19 gene. About another 25 to 35 percent of individuals with this disorder have identified mutations in the RPL5, RPL11, RPL35A, RPS7, RPS10, RPS17, RPS24, or RPS26 genes. In the remaining 40 to 50 percent of cases, the cause of the condition is unknown. Researchers suspect that other genes may also be associated with Diamond-Blackfan anemia.
106,903
What has replaced the term Near East?
Middle East
21,888
What causes Simple Kidney Cysts ?
The cause of simple kidney cysts is not fully understood. Obstruction of tubulestiny structures within the kidneys that collect urineor deficiency of blood supply to the kidneys may play a role. Diverticulasacs that form on the tubulesmay detach and become simple kidney cysts. The role of genetic factors in the development of simple kidney cysts has not been studied.
119,453
A crew in a spaceship travelling nearly the speed of light will not notice the change in what?
in the speed of time on board their vessel
61,884
Which nickname was given to the British sailors?
limeys
51,682
Since 2008, at least how many times has Microsoft released fixes for the RealTimeIsUniversal feature?
two
17,570
What does Utrecht provide for cyclist
Utrecht has an extensive network of cycle paths, making cycling safe and popular. 33% of journeys within the city are by bicycle
53,210
What is (are) After Surgery ?
After any operation, you'll have some side effects. There is usually some pain with surgery. There may also be swelling and soreness around the area that the surgeon cut. Your surgeon can tell you which side effects to expect. There can also be complications. These are unplanned events linked to the operation. Some complications are infection, too much bleeding, reaction to anesthesia, or accidental injury. Some people have a greater risk of complications because of other medical conditions. Your surgeon can tell you how you might feel and what you will be able to do - or not do - the first few days, weeks, or months after surgery. Some other questions to ask are - How long you will be in the hospital - What kind of supplies, equipment, and help you might need when you go home - When you can go back to work - When it is ok to start exercising again - Are they any other restrictions in your activities Following your surgeon's advice can help you recover as soon as possible. Agency for Healthcare Quality and Research
92,657
When did Boston withdraw its bid for hosting the Olympic games?
July 27, 2015
15,057
What influences role of minority leader?
personality and contextual factors
85,181
Who warned of impending Israeli attack on Syria in May 1967?
Soviet Union i
83,119
What is the most popular sport in the United Kingdom?
association football
79,744
What are the genetic changes related to alpha-methylacyl-CoA racemase deficiency ?
AMACR deficiency is caused by mutations in the AMACR gene. This gene provides instructions for making an enzyme called alpha-methylacyl-CoA racemase (AMACR). The AMACR enzyme is found in the energy-producing centers in cells (mitochondria) and in cell structures called peroxisomes. Peroxisomes contain a variety of enzymes that break down many different substances, including fatty acids and certain toxic compounds. They are also important for the production (synthesis) of fats (lipids) used in digestion and in the nervous system. In peroxisomes, the AMACR enzyme plays a role in the breakdown of a fatty acid called pristanic acid, which comes from meat and dairy foods in the diet. In mitochondria, AMACR is thought to help further break down the molecules derived from pristanic acid. Most individuals with AMACR deficiency have an AMACR gene mutation that results in a lack (deficiency) of functional enzyme. The enzyme deficiency leads to accumulation of pristanic acid in the blood. However, it is unclear how this accumulation is related to the specific signs and symptoms of AMACR deficiency.
93,974
Who supposedly encouraged the usage of Karnataka Brahmin priests at Pashupatinath Temple?
Adi Shankaracharya
87,466
What are riwaaydo?
popular musicals
23,156
What is (are) Osteopathia striata cranial sclerosis ?
Osteopathia striata cranial sclerosis is a type of skeletal dysplasia, which refers to a group of genetic conditions that affect the bones and hinder growth and development. The severity of the condition and the associated symptoms vary significantly from person to person, even within a single family. Features of the condition are generally present at birth and may include skeletal abnormalities (particularly at the ends of long bones), sclerosis (hardening) of the craniofacial bones, macrocephaly (unusually large head size), and characteristic facial features. Some affected people may also have developmental delay, hearing loss, heart defects and/or ophthalmoplegia (paralysis of the muscles surrounding the eyes). Osteopathia striata cranial sclerosis is caused by changes (mutations) in the WTX gene and is inherited in an X-linked dominant manner. Treatment is based on the signs and symptoms present in each person.
113,023
What causes Brittle diabetes ?
What causes brittle diabetes? There are multiple causes of brittle diabetes. Emotional stress seems to play an important role, in some cases leading to hormonal inbalances which can lead to brittle diabetes. Emotional stress can also lead to a shift in the behavior of an individual, leading them to neglect their self-care. Other cases can be traced to physiological causes, including malabsorption, delayed gastric emptying due to autonomic neuropathy (gastroparesis), celiac disease, impaired glucose counterregulation (which doesn't allow the patient's body to react as it should when blood glucose levels drop), hypothyroidism and adrenal insufficiency, drug or alcohol use, systemic insulin resistance, and abnormal insulin absorption or degradation.
99,943
What was the index published on December 18, 2008 lacking?
an accompanying Human Development Report
11,059
What Seattle fair lasts 24 days?
Seattle International Film Festival
35,979
What is the biggest use of asphalt?
road surfaces
37,144
How many students moved from Africa to Russia?
about 400,000
24,165
Who choreographed Chopiniana?
Michel Fokine
1,959
Where is Gustavo A. Madero?
North of the Historic Center
43,438
How many variations of the Old Testament offer the same meaning?
virtually every possible alternative reading
57,318
What did silt and marine fossils teach Shen Kuo?
geomorphology
53,904
Why are collaborative agreements important?
to explore the potential of new drug substances
36,793
Who attempted to assasinate Victoria while she was riding in a carriage?
Edward Oxford
37,722
What is (are) Cushing's syndrome ?
Cushing's syndrome is an endocrine disorder caused by prolonged exposure of the body's tissues to high levels of cortisol (a hormone produced by the adrenal gland). It most commonly affects adults between age 20 and 50 years. Signs and symptoms of Cushing's syndrome include upper body obesity, fatigue, muscle weakness, high blood pressure, backache, high blood sugar, easy bruising and bluish-red stretch marks on the skin. Affected women may also experience irregular menstrual periods and increased growth of body and facial hair. This condition may be caused by a variety of factors including long-term use of corticosteroid medications, tumors in the pituitary gland or adrenal adenomas.Treatment depends on the underlying cause, but may include decreasing the dosage of corticosteroids or surgery to remove tumors.
110,121
What decade started the push for Estonian independence?
1980s
63,773
How many people are affected by hystrix-like ichthyosis with deafness ?
HID is a rare disorder. Its prevalence is unknown.
107,297
What are the genetic changes related to renal tubular acidosis with deafness ?
Renal tubular acidosis with deafness is caused by mutations in the ATP6V1B1 or ATP6V0A4 gene. These genes provide instructions for making proteins that are parts (subunits) of a large protein complex known as vacuolar H+-ATPase (V-ATPase). V-ATPases are a group of similar complexes that act as pumps to move positively charged hydrogen atoms (protons) across membranes. Because acids are substances that can "donate" protons to other molecules, this movement of protons helps regulate the relative acidity (pH) of cells and their surrounding environment. Tight control of pH is necessary for most biological reactions to proceed properly. The V-ATPase that includes subunits produced from the ATP6V1B1 and ATP6V0A4 genes is found in the inner ear and in nephrons, which are the functional structures within the kidneys. Each nephron consists of two parts: a renal corpuscle (also known as a glomerulus) that filters the blood, and a renal tubule that reabsorbs substances that are needed and eliminates unneeded substances in urine. The V-ATPase is involved in regulating the amount of acid that is removed from the blood into the urine, and also in maintaining the proper pH of the fluid in the inner ear (endolymph). Mutations in the ATP6V1B1 or ATP6V0A4 gene impair the function of the V-ATPase complex and reduce the body's capability to control the pH of the blood and the fluid in the inner ear, resulting in the signs and symptoms of renal tubular acidosis with deafness.
96,764
What was Apple's highest quarterly profit as of Q1 2008?
$1.58 billion
2,727
When did Hyderabad become important culturally in India?
the mid-19th century
75,610
When the rule of law become an agenda item for the General Assembly?
1992
60,171
What are the symptoms of Alaninuria with microcephaly, dwarfism, enamel hypoplasia and diabetes mellitus ?
What are the signs and symptoms of Alaninuria with microcephaly, dwarfism, enamel hypoplasia and diabetes mellitus? The Human Phenotype Ontology provides the following list of signs and symptoms for Alaninuria with microcephaly, dwarfism, enamel hypoplasia and diabetes mellitus. If the information is available, the table below includes how often the symptom is seen in people with this condition. You can use the MedlinePlus Medical Dictionary to look up the definitions for these medical terms. Signs and Symptoms Approximate number of patients (when available) Abnormality of dental enamel 90% Aminoaciduria 90% Cognitive impairment 90% Incoordination 90% Intrauterine growth retardation 90% Microcephaly 90% Microdontia 90% Short stature 90% Type II diabetes mellitus 90% Autosomal recessive inheritance - Diabetes mellitus - Hypoplasia of dental enamel - Lactic acidosis - Severe short stature - The Human Phenotype Ontology (HPO) has collected information on how often a sign or symptom occurs in a condition. Much of this information comes from Orphanet, a European rare disease database. The frequency of a sign or symptom is usually listed as a rough estimate of the percentage of patients who have that feature. The frequency may also be listed as a fraction. The first number of the fraction is how many people had the symptom, and the second number is the total number of people who were examined in one study. For example, a frequency of 25/25 means that in a study of 25 people all patients were found to have that symptom. Because these frequencies are based on a specific study, the fractions may be different if another group of patients are examined. Sometimes, no information on frequency is available. In these cases, the sign or symptom may be rare or common.
110,480
What is the percentage of Muslims living in the Congo?
1.6%
9,753
Is oculodentodigital dysplasia inherited ?
Most cases of oculodentodigital dysplasia are inherited in an autosomal dominant pattern, which means one copy of the altered gene in each cell is sufficient to cause the disorder. In some cases, an affected person inherits the mutation from one affected parent. Other cases result from new mutations in the gene and occur in people with no history of the disorder in their family. Less commonly, oculodentodigital dysplasia can be inherited in an autosomal recessive pattern, which means both copies of the gene in each cell have mutations. The parents of an individual with an autosomal recessive condition each carry one copy of the mutated gene, but they typically do not show signs and symptoms of the condition. Fewer than ten cases of autosomal recessive oculodentodigital dysplasia have been reported.
108,434
In the Northern hemisphere's summer, what is the time difference between the UK and Chile?
five hours
17,394
What element is used in glass to keep it from becoming too hot?
Iron
66,569
What is (are) trisomy 13 ?
Trisomy 13, also called Patau syndrome, is a chromosomal condition associated with severe intellectual disability and physical abnormalities in many parts of the body. Individuals with trisomy 13 often have heart defects, brain or spinal cord abnormalities, very small or poorly developed eyes (microphthalmia), extra fingers or toes, an opening in the lip (a cleft lip) with or without an opening in the roof of the mouth (a cleft palate), and weak muscle tone (hypotonia). Due to the presence of several life-threatening medical problems, many infants with trisomy 13 die within their first days or weeks of life. Only five percent to 10 percent of children with this condition live past their first year.
104,286
Who is at risk for Childhood Extracranial Germ Cell Tumors? ?
Having certain inherited disorders can increase the risk of an extracranial germ cell tumor. Anything that increases your risk of getting a disease is called a risk factor. Having a risk factor does not mean that you will get cancer; not having risk factors doesnt mean that you will not get cancer. Talk with your childs doctor if you think your child may be at risk. Possible risk factors for extracranial germ cell tumors include the following: - Having certain genetic syndromes: - Klinefelter syndrome may increase the risk of germ cell tumors in the mediastinum. - Swyer syndrome may increase the risk of germ cell tumors in the testicles or ovaries. - Turner syndrome may increase the risk of germ cell tumors in the ovaries. - Having an undescended testicle may increase the risk of developing a testicular germ cell tumor.
116,018
What was the term for the oppositional literary sphere of a "multitude of versifiers and would-be-authors"?
Grub Street
33,966
What is (are) Inhalation Injuries ?
There are a variety of substances you can inhale that can cause acute internal injuries. Particles in the air from fires and toxic fumes can damage your eyes and respiratory system. They also can make chronic heart and lung diseases worse. Symptoms of acute inhalation injuries may include - Coughing and phlegm - A scratchy throat - Irritated sinuses - Shortness of breath - Chest pain or tightness - Headaches - Stinging eyes - A runny nose - If you already have asthma, it may get worse. The best way to prevent inhalation injuries is to limit your exposure. If you smell or see smoke, or know that fires are nearby, you should leave the area if you are at greater risk from breathing smoke. Environmental Protection Agency
117,647
For which part of the government is the MoD the headquarters?
British Armed Forces
22,306
How large is the Marshall Islands shark sanctuary in square miles?
772,000
20,149
What is subject to the Orphan Drug Act?
diseases involving fewer than 200,000 patients in the United States, or larger populations in certain circumstances
36,810
Who executed the apartheid?
the Arab government
24,065
What is the MSA of Raleigh?
1,214,516
51,491
Where was the earliest church labeled "Baptist" traced to?
Amsterdam
55,410
Along with the New Orleans VooDoo, Kansas City Brigade, Philadelphia Soul and Georgia Force, what former team returned for the 2011 season?
San Jose SaberCats
16,247
Is Laing distal myopathy inherited ?
This condition is inherited in an autosomal dominant pattern, which means one copy of the altered gene in each cell is sufficient to cause the disorder. In most cases, an affected person inherits the mutation from one affected parent. A small percentage of cases result from new mutations in the gene. These cases occur in people with no history of the disorder in their family.
98,585
What type of facility was the torch carried at instead.
stadium.
8,447
What country is listed as a republic?
Guinea-Bissau
21,297
What races are considered to be the of the the national races in Burma ?
national races such as Kachin, Kayah (Karenni), Karen, Chin, Burman, Mon, Rakhine, Shan, Kaman, or Zerbadee.
47,586
Is spondyloepimetaphyseal dysplasia, Strudwick type inherited ?
This condition is inherited in an autosomal dominant pattern, which means one copy of the altered gene in each cell is sufficient to cause the disorder.
93,735
Through what period did the second empire last?
1852 to 1870
74,810
Who is at risk for Peripheral Arterial Disease (P.A.D.)? ?
Smoking is the main risk factor for P.A.D. Your risk of P.A.D. increases four times if you smoke. Smoking also raises your risk for other diseases, such as coronary heart disease (CHD). On average, smokers who develop P.A.D. have symptoms 10 years earlier than nonsmokers who develop P.A.D. As you get older, your risk for P.A.D. increases, usually starting in your fifties. Older age combined with other risk factors, such as smoking or diabetes, also puts you at higher risk. African American men and women have a greater risk of developing P.A.D. than Caucasians. Your risk for P.A.D. is higher if you have diabetes, high cholesterol, high blood pressure, heart disease, or have had a stroke. A family history of these conditions also makes P.A.D. more likely.
118,679
What are the treatments for cerebrotendinous xanthomatosis ?
These resources address the diagnosis or management of cerebrotendinous xanthomatosis: - Gene Review: Gene Review: Cerebrotendinous Xanthomatosis - Genetic Testing Registry: Cholestanol storage disease These resources from MedlinePlus offer information about the diagnosis and management of various health conditions: - Diagnostic Tests - Drug Therapy - Surgery and Rehabilitation - Genetic Counseling - Palliative Care
95,921
Along with quick design times, what problem does Multiwire cut down on?
crosstalk
77,460
What is (are) congenital stromal corneal dystrophy ?
Congenital stromal corneal dystrophy is an inherited eye disorder. This condition primarily affects the cornea, which is the clear outer covering of the eye. In people with this condition, the cornea appears cloudy and may have an irregular surface. These corneal changes lead to visual impairment, including blurring, glare, and a loss of sharp vision (reduced visual acuity). Visual impairment is often associated with additional eye abnormalities, including "lazy eye" (amblyopia), eyes that do not look in the same direction (strabismus), involuntary eye movements (nystagmus), and increased sensitivity to light (photophobia).
104,936
What are the treatments for Adenoameloblastoma ?
How might adenoameloblastoma be treated? Treatment may require the removal of the legion as well as the surrounding tissues. Once the treatment is complete, recurrence of the legion is very rare.
98,987
New Delhi was ranked as the world's worst polluted city by what organization?
The World Health Organization
24,586
What is (are) Whooping Cough ?
Whooping cough is an infectious bacterial disease that causes uncontrollable coughing. The name comes from the noise you make when you take a breath after you cough. You may have choking spells or may cough so hard that you vomit. Anyone can get whooping cough, but it is more common in infants and children. It's especially dangerous for infants. The coughing spells can be so bad that it is hard for infants to eat, drink, or breathe. To make a diagnosis, your doctor may do a physical exam, blood tests, chest x-rays, or nose or throat cultures. Before there was a vaccine, whooping cough was one of the most common childhood diseases and a major cause of childhood deaths in the U.S. Now most cases are prevented by vaccines. If you have whooping cough, treatment with antibiotics may help if given early. Centers for Disease Control and Prevention
92,519
What is Hinkley point C
Hinkley Point C nuclear power station is a project to construct a 3,200 MW two reactor nuclear power station
48,813
For what environmental initiative is the city of New Haven known, primarily for instituting the first of its kind in America?
public tree planting program
44,038
Is congenital adrenal hyperplasia due to 11-beta-hydroxylase deficiency inherited ?
This condition is inherited in an autosomal recessive pattern, which means both copies of the gene in each cell have mutations. The parents of an individual with an autosomal recessive condition each carry one copy of the mutated gene, but they typically do not show signs and symptoms of the condition.
96,310
What are the treatments for prostate cancer ?
These resources address the diagnosis or management of prostate cancer: - American College of Radiology: Prostate Cancer Radiation Treatment - Genetic Testing Registry: Familial prostate cancer - Genetic Testing Registry: Prostate cancer, hereditary, 2 - MedlinePlus Encyclopedia: Prostate Brachytherapy - MedlinePlus Encyclopedia: Prostate Cancer Staging - MedlinePlus Encyclopedia: Prostate Cancer Treatment - MedlinePlus Encyclopedia: Prostate-Specific Antigen (PSA) Blood Test - MedlinePlus Encyclopedia: Radical Prostatectomy - MedlinePlus Health Topic: Prostate Cancer Screening - National Cancer Institute: Prostate-Specific Antigen (PSA) Test - U.S. Preventive Services Task Force These resources from MedlinePlus offer information about the diagnosis and management of various health conditions: - Diagnostic Tests - Drug Therapy - Surgery and Rehabilitation - Genetic Counseling - Palliative Care
93,796
What causes Nephrotic Syndrome in Adults ?
Nephrotic syndrome can be caused by diseases that affect only the kidneys, such as focal segmental glomerulosclerosis (FSGS) or membranous nephropathy. Diseases that affect only the kidneys are called primary causes of nephrotic syndrome. The glomeruli are usually the targets of these diseases for reasons that are not fully understood. In FSGSthe most common primary cause of nephrotic syndromescar tissue forms in parts of the glomeruli. In membranous nephropathy, immune molecules form harmful deposits on the glomeruli. Nephrotic syndrome can also be caused by systemic diseases, which are diseases that affect many parts of the body, such as diabetes or lupus. Systemic diseases that affect the kidneys are called secondary causes of nephrotic syndrome. More than 50 percent of nephrotic syndrome cases in adults have secondary causes, with diabetes being the most common.1
89,604
What Indian tribe was the Army's major campaign against?
Seminoles
33,041
What are the symptoms of Methionine adenosyltransferase deficiency ?
What are the signs and symptoms of Methionine adenosyltransferase deficiency? The Human Phenotype Ontology provides the following list of signs and symptoms for Methionine adenosyltransferase deficiency. If the information is available, the table below includes how often the symptom is seen in people with this condition. You can use the MedlinePlus Medical Dictionary to look up the definitions for these medical terms. Signs and Symptoms Approximate number of patients (when available) Intellectual disability 7.5% Autosomal dominant inheritance - Autosomal recessive inheritance - CNS demyelination - Dystonia - Hypermethioninemia - Hyperreflexia - Peripheral demyelination - The Human Phenotype Ontology (HPO) has collected information on how often a sign or symptom occurs in a condition. Much of this information comes from Orphanet, a European rare disease database. The frequency of a sign or symptom is usually listed as a rough estimate of the percentage of patients who have that feature. The frequency may also be listed as a fraction. The first number of the fraction is how many people had the symptom, and the second number is the total number of people who were examined in one study. For example, a frequency of 25/25 means that in a study of 25 people all patients were found to have that symptom. Because these frequencies are based on a specific study, the fractions may be different if another group of patients are examined. Sometimes, no information on frequency is available. In these cases, the sign or symptom may be rare or common.
100,244
Who was the father of Yazid ibn Abi Sufyan?
Abu Sufyan ibn Harb
36,974
Which of Homer's works did the last six books of the Aeneid connect to?
Iliad
20,531
Is Friedreich ataxia inherited ?
This condition is inherited in an autosomal recessive pattern, which means both copies of the gene in each cell have mutations. The parents of an individual with an autosomal recessive condition each carry one copy of the mutated gene, but they typically do not show signs and symptoms of the condition.
96,095
Installing keyloggers and making operating system modifications are examples of ways unauthorized users for what?
compromise security
27,193
Where is the FBI Academy?
Quantico, Virginia
30,546
What year did China and Japan reach a stalemate?
1941
78,321
To indicate something is feasible in Polish, what word could be used?
aktualny
80,430
What types of ions does typical glass have?
alkali and alkaline earth ions
66,638
Does botany only study plants?
included the study of fungi and algae
46,238
What sections of the Vedas did the Verdanta school favor?
latter parts
57,901
What is the outlook for Breast Cancer ?
Certain factors affect prognosis (chance of recovery) and treatment options.The prognosis (chance of recovery) and treatment options depend on the following: - The stage of the cancer (the size of the tumor and whether it is in the breast only or has spread to other parts of the body). - The type of breast cancer. - The age of the unborn baby. - Whether there are signs or symptoms. - The patients general health.
90,205
Where does a safari hunter usually stay?
tented camps
87,254
What North Carolina City hosts the Merlefest?
Wilkesboro
55,995
Kant's work continued to shape German thought and European philosophy well into what century?
20th
33,928
How many women's intercollegiate athletic teams are fielded by Northwestern?
11
39,603
What did this theological study do to set itself apart from the other groups ?
took its historical point of departure from Sophronios, Damascene, and their imitators.
81,421
How large is the Bronx?
42 square miles (109 km2)
86,007
On what Avenue is a southbound exit and entrance to the Harlem River Drive?
Park Avenue
21,616